Titlebook: Glial ? Neuronal Signaling; Glenn I. Hatton,Vladimir Parpura Book 2004 Springer Science+Business Media New York 2004 biology.brain.cell.ce

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Glutamate uptake by astroglia,nce that glutamate is similarly degraded following release. Instead, the actions of glutamate are ultimately terminated by uptake back into cells, a task accomplished primarily by a family of high affinity, Na.-dependent transporters (see review by Danbolt, 2001). Five different glutamate transporte

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Glutamate-mediated bi-directional signaling between neurons and astrocytes,tion [Ca.].. These evoked and/or spontaneous elevations of [Ca.]. in astrocytes can cause the release of a neurotransmitter, e.g., glutamate, from astrocytes, which signals to the presynaptic nerve terminal to modulate synaptic neurotransmission and/or released glutamate can affect postsynaptic cell

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The regulated release of transmitters from astrocytes, concluded that gap junctions mediate astrocyte-to-neuron signaling, Parpura et al. found evidence to support the notion that glutamate released from the astrocyte leads to an N-methyl-D-Aspartate receptor (NMDAR)-dependent elevation of neuronal calcium. Although additional support for the role of g

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Role of astrocytes in the formation, maturation and maintenance of synapses, — the astrocyte. As with other types of liaisons, the life of a synapse can be divided into three sequential phases: 1) the establishment of a physical contact, 2) a maturation process, which endows the synapse with its characteristic properties and 3) a stabilization or elimination phase, where on

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Work-Out: Holistic Active Management of NPL,s out the new boundaries of our knowledge regarding astrocyte-neuron interactions. It highlights the emerging reality that neuroscientists, whatever their areas of interest might be, must consider how glial cells fit into the picture. Ignoring this principle puts one at risk of getting the ‘story’ w

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Adaptability as Management Strategy,f direct sensory reception; rather, they receive information from specialized sensory cells, and/or from other neurons. In these cases, the sensory process has been replaced by a dendritic tree, which receives information via synaptic contacts. That is, dendrites are modified sensory processes adapt