Titlebook: Glaucoma Update III; Glaucoma Society of G. K. Krieglstein (Direktor der Universit?ts-Augen Conference proceedings 1987Latest edition Spri

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https://doi.org/10.1007/978-1-4614-0869-7ppears to be initiated at the scierai lamina cribrosa, just in front of the myelin line in the optic nerve head (Anderson and Hendrickson 1974; Minckler et al. 1978; Quigley and Green 1979; Quigley et al. 1981b). Physical rearrangements of the lamina occur early in the glaucoma damage process in hum

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Ceramic materials in aerospace,rent parts. In the prelaminar and laminar regions there is practically no myelin while behind the lamina cribrosa the axons become myelinated within the first mm. Myelinated nervous tissue can be expected to have blood flow and metabolic requirements that are lower than those of unmyelinated tissue

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https://doi.org/10.1007/978-1-4842-2271-3eak 26 mm Hg)..We have described (to be published) the typical angiographic features of senile sclerotic glaucoma. In this investigation we examined the fluorescein angiographic differences in patients with LTG with and without progressive visual field defects. The follow-up period for these patient

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https://doi.org/10.1007/978-3-030-71625-7 of a series of six normal, ocular hypertensive and glaucomatous patients with increasing risk for visual field loss, but with stable visual fields. Significant and borderline significant trends of changes of area of optic disc pallor were observed in three eyes associated with similar trends in ocu

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High Performance Structural Materialss with glaucoma, 85 ocular hypertensives and 20 normal controls. We also examined the prognostic value of the presence or absence of peripapillary atrophy in different diagnostic groups and the time dependence of the peripapillary atrophy area change. We measured the increase of the atrophy areas ov