Titlebook: Ghrelin in Health and Disease; Roy G. Smith,Michael O. Thorner Book 2012 Springer Science+Business Media New York 2012

凝乳

Ghrelin: Neuropeptide Regulator of Metabolism,he hypothesis that ghrelin primarily functions during negative energy balance to promote survival. Consistent with this idea, during calorie restriction, ghrelin increases blood glucose and suppresses glucose-stimulated insulin secretion from the pancreas. This important adaptive mechanism prevents

Embolic-Stroke

GOAT and the Regulation of Energy and Glucose Homeostasis, of energy homeostasis, especially adipogenesis. Recent data further suggest that GOAT is a significant factor in the regulation of glucose homeostasis as specific pharmacological GOAT inhibition improves glucose tolerance and insulin secretion. During caloric restriction, the ghrelin-GOAT system ma

設(shè)施

Ghrelin Regulation of Sleep, Circadian Clock, and Body Temperature, of the preproghrelin gene, particularly obestatin, are involved in maintaining normal body temperature and metabolism under conditions when increased metabolic heat production is required. Our understanding of the physiological role of the preproghrelin gene products expanded remarkably in the last

做事過(guò)頭

addict

套索

Use of Ghrelin and Ghrelin Receptor Agonists in Cancer- and Chemotherapy-Induced Cachexia,etics has not yet been fully established. In summary, more studies are needed looking into the long-term safety and efficacy of ghrelin and its mimetics in cancer patients and other chronic diseases associated with cachexia.

公豬

Itinerant

Ghrelin Suppression of Autoimmune Encephalomyelitis, In the C57BL/6 mouse model of EAE in which disease is induced by sensitization to myelin oligodendrocyte glycoprotein 35–55 peptide, we found that injections of ghrelin significantly reduced the clinical severity of EAE. The suppression of EAE was accompanied by a reduction in the mRNA levels of pr

Cholecystokinin

Physiological Roles of Des-Acyl Ghrelin,. Finally, recent results suggest that DAG has also proliferative and protective effects on β-cells. Transgenic mice over-expressing DAG display reduced fat mass and blood triglycerides. Also, the coadministration of AG and DAG reduces plasma FFA in GHD patients. Finally, results of recent prelimina

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