Titlebook: Genomics, Proteomics, and Clinical Bacteriology; Methods and Reviews Neil Woodford,Alan P. Johnson Book 2004 Humana Press 2004

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https://doi.org/10.1007/978-3-663-02161-2variety of newer technologies to identify the molecular target of the antibacterial agent. The advantage of this approach is that compounds already possess antibacterial activity, which is a property often challenging to engineer into molecules obtained from enzyme-based screening approaches. The re

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Die Arbeitsweise der Wechselstrommaschinenrial pathogens are very diverse, whereas others are genetically uniform, and some are, in essence, a single clone of a mother species that has been raised to species status due to the distinctiveness of the disease it causes (e.g., ., or .). The population structures of bacteria depend on the rate o

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https://doi.org/10.1007/978-3-658-01416-2l-subunit ribosomal RNA genes (rDNA) leading to the construction of branching trees representing the distance of divergence from a common ancestor has provided the mainstay of microbial phylogenetics. The approach has some limitations, particularly in the discrimination of closely related taxa, and

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https://doi.org/10.1007/978-3-658-07376-3y, through the identification of novel diagnostic targets and through the birth of a new discipline or “genomic epidemiology.” Current progress and future prospects for exploitation of genome sequences in clinical bacteriology are reviewed.

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