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Titlebook: Genome Editing in Cardiovascular and Metabolic Diseases; Junjie Xiao Book 2023 The Editor(s) (if applicable) and The Author(s), under excl

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樓主: arouse
31#
發(fā)表于 2025-3-27 00:52:05 | 只看該作者
32#
發(fā)表于 2025-3-27 02:38:11 | 只看該作者
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發(fā)表于 2025-3-27 05:22:00 | 只看該作者
https://doi.org/10.1007/978-981-10-6563-7ble to perform these types of experiments. Here, the different strategies for gene editing and their online tools are gathered, putting the light on its application in the study and treatment of cardiovascular and metabolic diseases.
34#
發(fā)表于 2025-3-27 10:41:09 | 只看該作者
Online Databases of Genome Editing in Cardiovascular and Metabolic Diseasesble to perform these types of experiments. Here, the different strategies for gene editing and their online tools are gathered, putting the light on its application in the study and treatment of cardiovascular and metabolic diseases.
35#
發(fā)表于 2025-3-27 17:08:51 | 只看該作者
Book 2023dology of genome editing. Genome editing is a genetic engineering technique precisely modified specific target genes of organism genome, which has been applied to basic theoretical research and production applications from plants and animals to gene therapy of human beings. Cardiovascular and metabo
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發(fā)表于 2025-3-27 19:30:59 | 只看該作者
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發(fā)表于 2025-3-28 00:38:52 | 只看該作者
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發(fā)表于 2025-3-28 02:17:55 | 只看該作者
E. Galán,R. Ca?ada,F. Fernández,B. Cerverasafety issues related to the once-and-done therapy focusing on CRISPR/Cas systems. We give an outlook on the potential gene targets prioritized by large-scale genetic studies of cardiovascular diseases and genome editing in precision medicine of dyslipidemia and atherosclerosis.
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發(fā)表于 2025-3-28 09:31:44 | 只看該作者
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發(fā)表于 2025-3-28 12:49:09 | 只看該作者
Genome Editing and Myocardial Developmentmodels are well-suited to study and predict clinical outcome. This review summarizes the anatomical and genetic timeline of myocardial development in both mice and humans, the potential of gene editing in typical CHD categories, as well as the use of mice thus far in reproducing models of human CHD and correcting the mutations that create them.
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