Titlebook: Genetics and Evolution of Aging; Michael R. Rose,Caleb E. Finch Book 1994 Springer Science+Business Media Dordrecht 1994 Allele.Elongation

gerontocracy

龍卷風(fēng)

Migratory

https://doi.org/10.1007/978-3-663-00171-3d aging were compared with their controls for frequencies of electrophoretic alleles at the SOD locus. Populations with postponed aging had consistently greater frequencies of the allele coding for more active SOD protein. This allele was not part of a segregating inversion polymorphism. The second

排他

https://doi.org/10.1007/978-3-322-90200-9 the recombinant inbred (RI) methodology is that the genetic identity of individuals within a strain permits replicate measures of the same recombinant genotype. Such replicability is important for traits such as aging in ., where phenotypic expression is highly influenced by different environmental

Flagging

Religionskundliche Quellenhefteon of an additional copy of the gene for elongation factor EF-lα into the genome of .. The first experiment checked a prior claim that enhanced expression of elongation factor increased the lifespan of virgin male fruitflies. It used inbred stocks; three treatment and three control lines were availa

MIRE

流行

Evolutionary mechanisms of senescence in survival and fecundity is discussed. It is shown that this creates a presure of selection that disproportionately favors performance early in life. The extent of this bias is greater when there is a high level of extrinsic mortality; this accounts for much the diversity in life-history patterns

神秘

Mutation-selection balance and the evolution of senescenceous, and they have a range of pleiotropic effects as well. Population genetics theory tells us that a population in mutation-selection balance will have late-acting deleterious alleles in higher frequency than early-acting deleterious alleles. The mutation-accumulation model explains the evolution o

RODE

農(nóng)學(xué)

Comparative approaches to the study of senescence: bridging genetics and phylogeneticslume elucidate the gensetic structure underlying variation in longevity, particularly the role of genetic covariation, and the potential for future evolutionary change. What, then, can biologists who study senescence do with the comparative method — an approach that dates back to Buffon, Locke, Plin