Titlebook: Genetic Engineering; Principles and Metho Jane K. Setlow Book 1992 Springer Science+Business Media New York 1992 Pathogene.cloning.genetic

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Regulation of Gene Expression by Thyroid Hormones and Retinoic Acid,e (T.) is the major circulating form and 3,5,3′-L-triiodothyronine (T.) its biologically more active metabolite, are synthesized . in all species of vertebrate organisms. Retinoids, in contrast, can be acquired only through dietary ingestion of retinol, β-carotene, or related compounds. Within speci

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Structural Constraints on Residue Substitution,l and natural selection at the level of protein structure and function. It is well established that the protein folding “code” is highly degenerate, in that many different sequences (for example, less than 20% identical) can adopt the same general fold. However, on analysis of homologous sequences i

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Molecular and Functional Analysis of the , Mating Type Genes of ,,rtile dikaryon. The monokaryon is the mycelium which develops from a single sexual spore. It has uninucleate cells and produces abundant asexual spores, the oidia. The dikaryon is formed by a compatible mating between two monokaryons. It has binucleate cells, one nucleus from each mate, and in ., ch

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Rudolf Steinbach,Horst Kohl,Gernot Krameritary, malignant and degenerative diseases. The success of this type of phenotype-based gene hunting has been dramatically illustrated through the cloning of genes responsible for Duchenne’s muscular dystrophy (1), cystic fibrosis (2) and neurofibromatosis type 1 (3), among many others.