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Titlebook: Genetic Control of the Susceptibility to Bacterial Infection; David E. Briles Book 1986 The Editor(s) (if applicable) and The Author(s), u

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31#
發(fā)表于 2025-3-26 21:53:18 | 只看該作者
, Gene Regulation of Mucosal Immunity and Susceptibility to , Infection in Mice,g of survivors, produced a strain of mice which displayed significantly increased resistance to the lethal effects of endotoxin. This finding suggested that the degree of sensitivity to endotoxin challenge was a heritable trait in mice. The demonstration by S. (1968) that inbred C3H/HeJ mice were re
32#
發(fā)表于 2025-3-27 03:21:45 | 只看該作者
Genetic Determination of Bacterial Virulence, with Special Reference to , ,osis, whose presence is noted to be correlated with virulence and whose loss, by mutation, etc., causes loss or great reduction in virulence in an experimental system. Logically any bacterial property indispensable for bacterial growth in the relevant compartment of the host, such as ability to grow
33#
發(fā)表于 2025-3-27 06:25:48 | 只看該作者
34#
發(fā)表于 2025-3-27 09:58:21 | 只看該作者
35#
發(fā)表于 2025-3-27 14:38:28 | 只看該作者
Genetic Control of Resistance to Mycobacterial Infection,e annual death rate from this disease reached 10% and over one-half of Indian families were eliminated in the first three generations. After 40 years and three generations, most susceptible individuals apparently died and the annual death rate had been reduced to 0.2% (. 1960). The presently high re
36#
發(fā)表于 2025-3-27 19:58:54 | 只看該作者
X-Linked Immune Deficiency (,) of CBA/N Mice,ked recessive gene (.) controlling unresponsiveness (Table 1). Recent mapping studies have located the . genes (or group of closely linked genes) between the tabby and hypophosphatemia genes on the X-chromosome (. et al. 1980). The large number of X-linked human immune deficiency diseases (. and . 1
37#
發(fā)表于 2025-3-28 00:23:09 | 只看該作者
, Gene Regulation of Mucosal Immunity and Susceptibility to , Infection in Mice,raced the chronology of mutational events which led to the defect in LPS responsiveness of C3H/HeJ mice. The unresponsiveness of C3H/HeJ mice to LPS was reported to be caused by a genetic mutation which occurred some time between 1960 and 1965 and was inbred into the C3H/HeJ line by 1968 (G. and R.
38#
發(fā)表于 2025-3-28 04:18:42 | 只看該作者
39#
發(fā)表于 2025-3-28 07:13:29 | 只看該作者
40#
發(fā)表于 2025-3-28 11:56:34 | 只看該作者
Gynecological Applications of Cytology,ked recessive gene (.) controlling unresponsiveness (Table 1). Recent mapping studies have located the . genes (or group of closely linked genes) between the tabby and hypophosphatemia genes on the X-chromosome (. et al. 1980). The large number of X-linked human immune deficiency diseases (. and . 1
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