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Titlebook: Genetic Causes of Cardiac Disease; Jeanette Erdmann,Alessandra Moretti Book 2019 Springer Nature Switzerland AG 2019 induced pluripotent s

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發(fā)表于 2025-3-21 18:09:50 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Genetic Causes of Cardiac Disease
編輯Jeanette Erdmann,Alessandra Moretti
視頻videohttp://file.papertrans.cn/383/382460/382460.mp4
概述Presents the recent developments in the field of cardiac disease genetics..Different models for study and analysis are discussed, including iPSC..Written and edited by leading figures in the field..Wr
叢書名稱Cardiac and Vascular Biology
圖書封面Titlebook: Genetic Causes of Cardiac Disease;  Jeanette Erdmann,Alessandra Moretti Book 2019 Springer Nature Switzerland AG 2019 induced pluripotent s
描述.This book provides a comprehensive summary of the latest developments in the field of the genomics of cardiac disease.?Written and edited by leading clinicians and scientists involved in the analysis and therapy of genetic cardiac disorders, it discusses the genetic causes of a variety of cardiac diseases, such as the complex genetics and etiology of congenital heart diseases. It also explores sex differences in prevalent diseases, genetics-based therapeutic strategies and the use of various animal models and alternatives...The book is intended for research scientists and clinical scientists in the cardiovascular field, human geneticists and cardiologists..
出版日期Book 2019
關鍵詞induced pluripotent stem cells, iPSC; genetics; cardiac diseases; mouse models; zebrafish; complex geneti
版次1
doihttps://doi.org/10.1007/978-3-030-27371-2
isbn_softcover978-3-030-27373-6
isbn_ebook978-3-030-27371-2Series ISSN 2509-7830 Series E-ISSN 2509-7849
issn_series 2509-7830
copyrightSpringer Nature Switzerland AG 2019
The information of publication is updating

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沙發(fā)
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地板
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Controlling im Gesundheitswesen risk factors are a leading cause of death and morbidity in the world [1–3]. According to World Health Organization estimates, CVDs are responsible for 151,377 million disability-adjusted life years (DALY). DALY is the number of years lost because of ill health, disability or premature death. Corona
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發(fā)表于 2025-3-22 10:49:56 | 只看該作者
https://doi.org/10.1007/978-3-658-45895-9ew recent findings on the genetic basis and inheritance patterns of CHD. Rather than provide lists of identified genes, instead we offer a conceptual framework to understand the relationship between genetic variation and CHD. We review recent studies utilizing contemporary techniques, some of which
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https://doi.org/10.1007/978-3-7091-6219-40 people [1–3]. Inherited in an autosomal-dominant fashion, it is associated with lifelong, severe elevations in low-density lipoprotein-cholesterol (LDL-c) levels. Individuals with FH have a markedly elevated risk of premature ischemic heart disease, 5–20-fold higher than the general population [4–
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Edmond Irani Liu,Matthias Althoffd. This includes tens of thousands of RNAs sized larger than 200 nucleotides that are not translated into proteins and are referred to as long noncoding RNAs (lncRNAs). Until now, only a few of these lncRNAs have been functionally characterized. Here we highlight lncRNAs related to cardiovascular ph
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發(fā)表于 2025-3-22 22:36:16 | 只看該作者
Corona und mediale ?ffentlichkeitenere with these devastating diseases is limited. Cardiomyopathies, which are mainly due to genetic causes, make a significant portion of heart failure. Current pharmacological treatment of cardiovascular diseases focuses on symptoms rather than the underlying cellular mechanisms, and the gaps in our
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Petter Gottschalk,Christopher Hamertoncells (hiPSCs) from diseased and healthy individuals, the discovery of the clustered regularly interspaced short palindromic repeats (CRISPR)/CRISPR-associated 9 (Cas9) genome editing system, and the possibility to identify disease-related genetic variants in human populations in genome-wide associa
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