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Titlebook: Genes and Environment in Cancer; Manfred Schwab,Hartmut M. Rabes,Hans Peter Hofschn Conference proceedings 1998 Springer-Verlag Berlin · H

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書目名稱Genes and Environment in Cancer
編輯Manfred Schwab,Hartmut M. Rabes,Hans Peter Hofschn
視頻videohttp://file.papertrans.cn/383/382408/382408.mp4
叢書名稱Recent Results in Cancer Research
圖書封面Titlebook: Genes and Environment in Cancer;  Manfred Schwab,Hartmut M. Rabes,Hans Peter Hofschn Conference proceedings 1998 Springer-Verlag Berlin · H
描述Different cancer types can result from a multiplicity of genetic and environmental factors. In recent years a number of genes have been identified as strong determinants for particular forms of cancer (particularly colon and breast cancer). The incomplete penetrance often evinced by the mutations of these genes has raised the possibility that additional endogenous or exogenous determinants contribute to cancer development or suppression. The major aim of this book is to present an integrated view of the various environmental, epidemiological and genetic determinants that contribute to a disease syndrome collectively known as "cancer".
出版日期Conference proceedings 1998
關(guān)鍵詞DNA; Genetik; Termination; breast cancer; cancer; cancer therapy; carcinogenesis; carcinoma; chromosome; colo
版次1
doihttps://doi.org/10.1007/978-3-642-46870-4
isbn_softcover978-3-642-46872-8
isbn_ebook978-3-642-46870-4Series ISSN 0080-0015 Series E-ISSN 2197-6767
issn_series 0080-0015
copyrightSpringer-Verlag Berlin · Heidelberg 1998
The information of publication is updating

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Molecular Epidemiology of Human Cancertro and in vivo laboratory models, biochemistry and epidemiology to infer individual cancer risk. Molecular dosimetry of carcinogen exposure is an important facet of molecular epidemiology and cancer risk assessment. Carcinogen macromolecular adduct levels, cytogenetic alterations and somatic cell m
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Molecular Epidemiology of Environmental Carcinogenesisiefly reviews molecular evidence that two types of susceptibility factors — common predisposing genetic traits and young age at exposure — convey heightened risk from certain exposures. Examples are drawn from molecular epidemiologic studies of common environmental carcinogens such as polycyclic aro
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Molecular Basis of DNA Repair Mechanisms and Syndromes in the nucleotide sequence varying from point mutations to gross chromosomal aberrations which can alter the expression or functioning of genes implicated in regulation of cell proliferation and differentiation, thereby forwarding the cell in the multistep process of carcinogenesis. To prevent thes
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