Titlebook: Generalized Non-Convulsive Epilepsy: Focus on GABA-B Receptors; C. Marescaux,M. Vergnes,R. Bernasconi Conference proceedings 1992 Springer

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Non-opioid Analgesics in Dental Practice, regions studied over the concentration range 25 nM to 400 nM. This indicated that there was no change in either the Kd (affinity) or Bmax (receptor number) in these animals and that the pharmacological basis for the efficacy of GABA. antagonists in this seizure condition probably lies elsewhere.

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GABA receptors in rats with spontaneous generalized nonconvulsive epilepsy, regions studied over the concentration range 25 nM to 400 nM. This indicated that there was no change in either the Kd (affinity) or Bmax (receptor number) in these animals and that the pharmacological basis for the efficacy of GABA. antagonists in this seizure condition probably lies elsewhere.

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Pharmacological models of generalized absence seizures in rodents, low dose pentylenetetrazole, penicillin, THIP and AY-9944. All of these models share behavioral and EEG similarity to human absence seizures and show pharmacologic specificity for antiabsence drugs such as ethosuximide and trimethadione. Moreover, the absence seizures induced by these agents are ex

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,Genetic absence epilepsy in rats from Strasbourg — A review,ent recurrent generalized non-convulsive seizures characterized by bilateral and synchronous spike-and-wave discharges accompanied with behavioural arrest, staring and sometimes twitching of the vibrissae. Spontaneous SWD (7–11 cps, 300–1,000 μV, 0.5–75 sec) start and end abruptly on a normal backgr

潛伏期

Cortical and thalamic lesions in rats with genetic absence epilepsy,ous cortical and thalamic lesions on the occurrence of spontaneous SWD was examined in rats from a strain with genetic absence epilepsy..Cortical ablations suppressed SWD recorded in the thalamus. KCl induced unilateral cortical spreading depression and transiently suppressed SWD in the ipsilateral