Titlebook: Generalized Epilepsy; Neurobiological Appr Massimo Avoli,Pierre Gloor,Robert Naquet Book 1990 Birkh?user Boston, Inc. 1990 PET.brain.cortex

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Pharmacology of the Inhibitory Systems in Primary Generalized Epilepsy of “Petit Mal” Typered evident to us about 10 years ago while we were studying the effect of homotaurine (3-aminopropanesulfonic acid, 3APS), a highly specific GABA agonist, on various seizure models, in view of its potential use as an antiepileptic agent. An intravenous injection of 3APS in cats with an acute neocor-

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Role of Neurotransmitters in the Genesis of Epileptiform Dischargesuncontrolled, paroxysmal depolarization and firing if indiscriminate excitation was not constantly opposed by very potent control mechanisms. The remarkable ability of the normal cortex to function for many years without any signs of major uncontrolled activity is irrefutable evidence of an astonishingly effective regulation of firing.

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Thalamocortical Relationships in Generalized Epilepsy with Bilaterally Synchronous Spike-and-Wave Dial EEG background activity has always made attractive the hypothesis that some central pacemaker located in the depths of the brain and projecting diffusely to the cortex was responsible for the origin of SW discharges (Jasper and Kershman, 1941; Penfield and Jasper, 1946, 1954); see also review in Gloor, 1978).

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topic. These reviews, however, have focused almost exclusively on studies performed in models of partial (or focal) epilepsy. It was natural that scientists interested in the fundamental mechanism of epileptogenesis at the cellular level were attracted to work on models in which the epileptogenic dy

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Argument as Conflict: Then and Now,uncontrolled, paroxysmal depolarization and firing if indiscriminate excitation was not constantly opposed by very potent control mechanisms. The remarkable ability of the normal cortex to function for many years without any signs of major uncontrolled activity is irrefutable evidence of an astonishingly effective regulation of firing.

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The Superpowers and the October Waral EEG background activity has always made attractive the hypothesis that some central pacemaker located in the depths of the brain and projecting diffusely to the cortex was responsible for the origin of SW discharges (Jasper and Kershman, 1941; Penfield and Jasper, 1946, 1954); see also review in Gloor, 1978).