Titlebook: Gene Expression in Muscle; Richard C. Strohman,Stewart Wolf Book 1985 The Editor(s) (if applicable) and The Author(s), under exclusive lic

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https://doi.org/10.1007/978-981-16-4534-1d into two main groups: 1. Deficiencies of enzymes that occur in single molecular forms which are already present in fetal tissues and continue to be synthesized throughout life. Defects of these “household enzymes” usually cause functional impairment of muscle, as well as other organs and tissues.

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l, histochemical, and biochemical methods have demonstrated clear dystrophic abnormalities in the posterior latisslmus dorsi (PLD) while the anterior latissimus dorsi (ALD), a slow tonic muscle, is unaffected by the disease (216,219). Most of this research has been performed on normal and dystrophic

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https://doi.org/10.1007/978-981-13-0101-8ract (EE). However, they do not grow, when incubated with a medium composed of Eagle’s MEM and horse serum (basal medium). This indicates that EE contains essential substance(s) for chick myogenic cell growth, which is contained in neither the artificial medium nor horse serum.

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muscle fibers (367, 368) with different physiological and metabolic properties. For example, fast and slow twitch fibers contain both different myosin heavy chain and myosin light chain complements; and among the fast twitch fibers, IIa and IIb fibers contain myosins with qualitatively distinct myos

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