Titlebook: Gangliosides and Neuronal Plasticity; Guido Tettamanti,Robert W. Ledeen,Gino Toffano Book 1986 Springer-Verlag Berlin Heidelberg 1986 Germ

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書目名稱Gangliosides and Neuronal Plasticity影響因子(影響力)學科排名




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Annals of Industrial Engineering 2012on structural features of the analyte even if it is present only in minute quantities. Today, its application to the analysis of trace amounts of material is practically indispensable in biological and medical sciences (Burlingame, 1985).

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insincerity

Fortuitous

The Investigation of Mortality,t al., 1971; Cuatrecasas, 1973), viruses (Haywood, 1974; Holmgren et al., 1980), hormones (Mullin et al., 1976) and chemical mediators (Woolley and Gommi, 1965). On the other hand, some special gangliosides have been proposed to be regulators of cell growth or differentiation (Bremer et al., 1984; T

發(fā)電機

發(fā)電機

Annals of Life Insurance Medicine 5unds in tissue sections. Proper interpretation of these immunological data requires knowledge of certain principles of carbohydrate immunochemistry and these will be reviewed in the next section. Some recent immunocytochemical studies will be presented in the final section.

intimate

Annals of Life Insurance Medicine 6hese studies was that predominance of GD3 may characterize mitotically active precursor cells (Dreyfus et al., 1975; R?sner, 1980; 1982; R?sner et al., 1985; Goldmann et al., 1984) and accretion of more complex gangliosides indicates the transition to postmitotic young neurons (R?sner, 1980; 1982).

征稅

Joachim Yahalom M.D.,Subhash Gulatiant in the nervous system (Ledeen and Yu, 1982; Ledeen, 1983; Ando, 1983; Ledeen, 1985). Gangliosides comprise a major part of the glycoconjugate network extending from the neuronal membrane surface. Recent investigations suggest that gangliosides may exert a critical role in the regulation of cell

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https://doi.org/10.1007/978-3-662-61755-7tal stage, and then may deteriorate in their function in senescence. Giacobini emphasized the relevance of reduced synaptic transmission to malfunction of the aging brain. Agerelated alterations of synaptic functions have been reported in terms of the levels, synthetic rates and releasing activities