Titlebook: Galectins; Methods and Protocol Sean R. Stowell,Connie M. Arthur,Richard D. Cummin Book 2022Latest edition Springer Science+Business Media,

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青石板

https://doi.org/10.1007/978-1-4614-3609-6an structures, and functional studies have indicated that a family of glycan-binding proteins, galectins, can significantly modulate lymphocyte development and function by interacting with these glycans. Several galectins have a varying degree of affinity for the .acetyllactosamine (LacNAc) disaccha

欄桿

Air Evacuation of the Neurosurgical Patientcted mutagenesis of the hydrophilic residues His, Asn, Arg, and Glu, involved in carbohydrate recognition, abolished the binding affinity of the derived mutants to β-galactosides, whereas only N46A caused increased affinity to GalNAcα1-3Gal-containing oligosaccharides and loss of β-galactoside-bindi

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Elizabeth Bridges,Melissa A. Buzbee-Stilesrresponding engineering of galectin-specific cDNA will answer questions on the fate of a signal peptide-bearing protein variant after its entry into the endoplasmic reticulum (ER). Affinity chromatography and mass-spectrometric analysis of occupancy of potential N-glycosylation sites for the galecti

群島

First Helicopters and Rotor Systems and are considered as not only biomarkers of human diseases but also molecular targets for treating cancer and inflammatory illnesses in many organs. In the glycobiology field, it is crucial to determine the pattern of galectin expression and location in cells and tissues. Confocal microscopy is a

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Anomalies in the Regulatory Structure,atus of glycan branching or chain length (e.g., core 1 vs core 2 mucin-type O-glycans and polyLacNAc additions) as well as of sialylation/sulfation has been delineated to convey signals. They are “read” by galectins, for example regulating lattice formation on the membrane and cell growth. Owing to

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Anthrp

Aeronomy of the Middle Atmosphereally exposed glycans and accumulate around endocytic vesicles or organelles damaged by various disruptive substances. Accumulated galectins engage other cytosolic proteins toward damaged vesicles, leading to cellular responses, such as autophagy. Disruptive substances include bacteria, viruses, part

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https://doi.org/10.1007/978-1-0716-2055-7oxidative inactivation; thermodynamics; glycan microarrays; neoplastic disease; cancer cells

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