Titlebook: GM3 Signaling; Cheorl-Ho Kim Book 2020 Springer Nature Singapore Pte Ltd. 2020 Gangliosde.GM3.Sialic acid.sugar code.Anti-Tumor.EGFR-GM3 i

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,GM3 Interacts with TGFβRs in the Epithelial–Mesenchymal Transition (EMT) During Posterior Capsular [2]. The lens epithelial cells are transdifferentiated to mesenchymal-like cells such as myofibroblasts with the phenotypes such as membrane presence of fibronectin, type 1 collagen, and α-smooth muscle actin (SMA) [2, 3]. PCO is frequently caused by the EMT process in lens epithelial cells through

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Galectin-1 Promotes Tumor Growth and Escapes Immune Surveillance,ognition receptors (PRRs) for self-recognition vs. non-self-recognition. The Janeway and Medzhitov model shows that PRRs recognize pathogens through MAMPs, because MAMPs are absent from the host. The paradox of galectins, which bind similar “self” and “non-self” patterns, is raised due to limited kn

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GM3-HGFR, FGFR, and PDGFR Cancer Cell Behavior and IGF-1R in Diabetic Wound Healing,se of GFRs. GM3 is a precursor substrate for biosynthesis of the gangliosides of complex types including a, b, and c series. The precise mechanism(s) responsible for GM3 engagement is not clear in the malignant transformation, where GM3 exerts anti-tumor actions or promoting actions on tumor behavio

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GM3, Caveolin-1 and Insulin Receptor in Insulin Resistance,iabetic role of GM3 has been attributed to impaired insulin action [4]. In human studies, aberrantly increased serum level of GM3 is detected in hyperglycemic, hyperlipidemic, and T2DM patients [5, 6]. Glucocerebrosidase deficient Gaucher disease shows insulin resistance with serum GM3 level [7]. Th

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Book 2020ing inflammation, inhibiting tumor angiogenesis and tumor growth, and suppressing arthritis arehighlighted, and attention drawn to the significance of GM3 as a driver of impaired wound healing in diabetics. The book will be of interest to all who want a comprehensive update on research in this field

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https://doi.org/10.1007/978-3-319-02744-9)-reactive reagent. He, for the first time, called this glycolipid isolated from horse erythrocytes as hematoside in order to distinguish this glycolipid hematoside from the ganglioside isolated by Klenk’s preceding report. However, in fact, the hematoside is GM3 [4]. The hematoside isolated by Yama

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https://doi.org/10.1007/978-981-99-5362-2 2]. They are together with allogenic sphingolipids in raft microdomains. Ganglioside is also pivotal in central myelination of neuronal responses. Cell-type expression of gangliosides and diversity may solve the clues what kinds of gangliosides interact with their proteins and how do interact? Form

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