| 書目名稱 | Free Radicals and Inflammation | | 編輯 | Paul G. Winyard,David R. Blake,Christopher H. Evan | | 視頻video | http://file.papertrans.cn/348/347965/347965.mp4 | | 叢書名稱 | Progress in Inflammation Research | | 圖書封面 |  | | 描述 | As in so many fields of scientific endeavour following the molecular biology revo- lution, our knowledge of the role of radicals not only in pathological states, but in basic physiology has developed exponentially. Indeed, our evolving concepts have, like so many political parties, been forced into dramatic "V-turns" and contortions. Within our working lives, we have had to debate whether radicals made any con- tribution to any pathology, whilst now it is difficult not to entertain the view that every physiological process is pivotally controlled by exquisitely sensitive radical reactions. Inflammation is, of course, an example of pathology evolving from physiology, and in this book we have called upon both scientists and clinicians who have research interests in the complex switching mechanisms that sustain these transi- tions. The book as a whole explores, from a physiological standpoint, how deter- ministic radical systems sensitive to their initial conditions can interdigitate, iterate and feed back to control diverse cellular processes that create the inflammatory response. Whilst systems such as these to a mathematician would provide the basis for a chaotic response, one is f | | 出版日期 | Book 2000 | | 關鍵詞 | Arthritis; apoptosis; biology; cell; cell death; diseases; inflammation; nitrogen; rheumatism; rheumatoid art | | 版次 | 1 | | doi | https://doi.org/10.1007/978-3-0348-8482-2 | | isbn_softcover | 978-3-0348-9586-6 | | isbn_ebook | 978-3-0348-8482-2Series ISSN 1422-7746 Series E-ISSN 2296-4525 | | issn_series | 1422-7746 | | copyright | Springer Basel AG 2000 |
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