Titlebook: Experimental and Clinical Neuropathology; Proceedings of the F Kurt Jellinger (Director),Filippo Gullotta,Mirosla Conference proceedings 19

輕快帶來危險

Effect of Prolonged Acetylcholinesterase Inhibition on Postnatal Brain Development in Rabbit I. Levecaused the increase of serotonin concentration in all brain regions. After prolonged intoxication the decrease of serotonin concentration in brain stem, mesencephalon and hippocampus for 78, 74, 41% respectively was observed. The cholinergic-serotonergic link is discussed.

勤勞

飛來飛去真休

鋪子

Exonerate

The Permeability of the Blood-Brain Barrier in Acute Hypertension. Comparison of an Endogenous and Esec or 30 min the animals were killed by perfusion fixation with 8% formaldehyde perfusion..The barrier passage of exogenous HRP (75 mg i.v.) given 10 min before killing (5 rats) and the passage of endogenous anti-HRP antibodies of the IgG class (8 rats, produced by antigenic stimulation beforehand)

是限制

隱語

The Role of Lactic Acidosis in the Ischemic Nerve Cell Injurygroup of rats were fasting (f-rats), while the other was infused with glucose before induction of the ischemia (g-rats). In f-rats the lactate accumulating in the cerebral cortex was about 15 μmol · g., whereas in g-rats it rose to about 35 μmol · g.. In f-rats considerable recovery of the energy st

蟄伏

Acceleration Induced Head Injury in the Monkey. I. The Model, Its Mechanical and Physiological Correock tester subjects a monkey to a non-impact controlled single sagittal rotation which displaces the head 60° in 10–20 msec. Results derived from 53 experiments show that a good correlation exists between acceleration delivered to the head, the resultant neurological status and the brain pathology.

Crohns-disease

Fraudulent

Glucose Utilization During Chromatolysis: a 14C Deoxyglucose Studyowed increased radioactivity in the motor nuclei of the operated side between land 28 days post operation. This suggests an increase of glucose utilization and thus an enhanced energy supply to the facial and hypoglossal nuclei during the process of axonal reaction.