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Titlebook: Experimental Hepatocarcinogenesis; M. B. Roberfroid,V. Préat Conference proceedings 1988 Plenum Press, New York 1988 DNA.apoptosis.biology

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樓主: 挑染
31#
發(fā)表于 2025-3-27 00:21:55 | 只看該作者
32#
發(fā)表于 2025-3-27 01:49:25 | 只看該作者
33#
發(fā)表于 2025-3-27 08:14:45 | 只看該作者
Structure and Organization of Virus Genomes,ation. This is apparently true for the development of all kinds of malignant tumors, induced by physical, chemical or viral factors, and applies also to hepatocarcinogenesis. This contribution is aimed at dissecting some critical phases of the carcinogenic process in the liver with respect to cell proliferation.
34#
發(fā)表于 2025-3-27 11:38:45 | 只看該作者
35#
發(fā)表于 2025-3-27 17:03:43 | 只看該作者
Genome Mapping and Genomics in Animalstment, the modulation is considered as positive whereas a treatment which decreases the incidence and/or the yield of cancer and/or increases the latency period is considered as a negative modulator. (Roberfroid, 1987)
36#
發(fā)表于 2025-3-27 18:34:02 | 只看該作者
Modulating Factors of Hepatocarcinogenesistment, the modulation is considered as positive whereas a treatment which decreases the incidence and/or the yield of cancer and/or increases the latency period is considered as a negative modulator. (Roberfroid, 1987)
37#
發(fā)表于 2025-3-28 00:23:22 | 只看該作者
Hepatocarcinogens different chemical nature. In experimental animals, mostly the rat and the mouse, quite a number of diverse chemicals have been demonstrated to produce liver tumors. Accordingly, such chemicals are called hepatocarcinogens.
38#
發(fā)表于 2025-3-28 06:00:16 | 只看該作者
39#
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