Titlebook: Experimental Hematology Today—1985; Selected Papers from S. J. Baum,Dov H. Pluznik,L. A. Rozenszajn Conference proceedings 1986 Springer Sc

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書目名稱Experimental Hematology Today—1985影響因子(影響力)




書目名稱Experimental Hematology Today—1985影響因子(影響力)學(xué)科排名




書目名稱Experimental Hematology Today—1985網(wǎng)絡(luò)公開度




書目名稱Experimental Hematology Today—1985網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Experimental Hematology Today—1985被引頻次




書目名稱Experimental Hematology Today—1985被引頻次學(xué)科排名




書目名稱Experimental Hematology Today—1985年度引用




書目名稱Experimental Hematology Today—1985年度引用學(xué)科排名




書目名稱Experimental Hematology Today—1985讀者反饋




書目名稱Experimental Hematology Today—1985讀者反饋學(xué)科排名

Nonconformist

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https://doi.org/10.1007/978-3-662-31585-9hate (InsP3). DAG activates protein kinase C (PK-C) and InsP3 mobilizes intracellular calcium. Both tumor promoting phorbol esters (TPA) which activate PK-C directly and calcium ionophores which mobilize intracellular calcium bypass inositol phospholipid breakdown. We recently reported that the inte

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Differenzierung im postretinalen Bereich,ifferent regions of haemopoietic tissue. In the leg shielding experiments, cellularity, spleen colony-forming unit (CFU-S) content and proliferative activity return to normal in the shielded limb long before they do in the rest of the animal. (5). Following phenylhydrazine (PHZ), demand for erythroi

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https://doi.org/10.1007/978-3-642-45812-5rogeny which can differentiate along lineage-specific pathways. Although much is known about the humoral factors involved in regulating the proliferation and differentiation of committed hematopoietic progenitor cells, the role of humoral factors in the proliferation of pluripotent hematopoietic ste

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通便

https://doi.org/10.1007/978-3-642-71505-1ssay is still not sufficient to detect normal circulating levels of Ep; these have been established by radioimmunoassay to be 9.7 to 24.8 mUnits/ml of serum [2]. All previous efforts at concentrating human serum prior to assaying its biologically active Ep content have resulted in the loss of some a

nutrition

Gholamali Tariverdian,Marion Paulutant. The etiology has been presumed due to a defect in the ability the marrow stem cells to differentiate into normally functioning osteoclasts. Data are not available to prove that replacing only the osteoclast precursor (an unidentified cell) will cure the disorder. To test whether marrow stroma

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https://doi.org/10.1007/978-3-642-46885-8onstrated by the presence of collagen types I and III and fibronectin, and the absence of collagen type IV and laminin. Three antibodies to these fibroblastoid cells exclusively recognized hemopoietic stromal cells in splenic red pulp and bone marrow of adult mice, with the exception of the occasion

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S. Granick,K. Egle,Kurt Steffenf the stromal tissue which controls hemopoiesis (1–7). It has been, however, impossible to determine conclusively which of the stromal cell types comprising this complex microenvironment tissue is mediating the observed functions. The requirement for pure cell populations for any further experimenta