Titlebook: Excitotoxins; Proceedings of an In Kjell Fuxe,Peter Roberts,Robert Schwarcz Conference proceedings 1983 Springer Science+Business Media New

可憐

書目名稱Excitotoxins影響因子(影響力)




書目名稱Excitotoxins影響因子(影響力)學(xué)科排名




書目名稱Excitotoxins網(wǎng)絡(luò)公開度




書目名稱Excitotoxins網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Excitotoxins被引頻次




書目名稱Excitotoxins被引頻次學(xué)科排名




書目名稱Excitotoxins年度引用




書目名稱Excitotoxins年度引用學(xué)科排名




書目名稱Excitotoxins讀者反饋




書目名稱Excitotoxins讀者反饋學(xué)科排名

BOLT

ovation

The Role of Seizures in Kainic Acid Induced Brain Damageental conditions the distant damage is largely due to the spread of paroxysmal discharge and not to a direct action of the toxin (by diffusion). The usefulness of KA to study the relationship between epilepsy and brain pathology is reviewed and the possible causes of the particular vulnerability of the ammon’s horn to epilepsy is discussed.

Ordnance

指數(shù)

Mechanisms of Excitotoxins Examined in Organotypic Cultures of Rat Central Nervous Systemle et al, 1977; Fibiger, 1978). Our studies (Whetsell et al, 1979; Whetsell, 1979; Whetsell and Nicklas, 1980) in organotypic cultures of rat striatum (CA), striatum-plus-frontal cortex (CA-CX) and frontal cortex alone (CX), have demonstrated that the neurotoxic effects of KA can be observed only in CA-CX and CX cultures.

initiate

https://doi.org/10.1007/978-3-663-07712-1 tasks in the brain. It is involved in energy metabolism and fatty acid synthesis, incorporated into proteins and peptides and is even a precursor for the inhibitory neurotransmitter GABA. In addition its action as a neuroexcitant has been regarded as universal and non-specific.

initiate

, and Forward Lease ,—Case Study,o alterations in glial cells secondary to massive depolarization of neurons. These results and those of others are discussed in terms of their implication for the mechanism of neurotoxicity of excitatory substances.

侵略者

Asymptomatic

https://doi.org/10.1007/978-3-030-33380-5t which depresses agonist-induced responses in the order kainate > quisqualate > N-methyl-D- aspartate > L-glutamate. Other sulphonic peptides have similar, though weaker actions. The actions of excitatory amino acid antagonists in depressing polysynaptic and monosynaptic excitation in the spinal cord are described.

Cholecystokinin

Recent Advances in the Pharmacology of Excitatory Amino Acids in the Mammalian Central Nervous Systet which depresses agonist-induced responses in the order kainate > quisqualate > N-methyl-D- aspartate > L-glutamate. Other sulphonic peptides have similar, though weaker actions. The actions of excitatory amino acid antagonists in depressing polysynaptic and monosynaptic excitation in the spinal cord are described.