Titlebook: Excitatory Amino Acids; Peter J. Roberts,Jon Storm-Mathisen,Harry F. Bradf Book 1986 The Editors and the Contributors 1986 amino acid.huma

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書目名稱Excitatory Amino Acids影響因子(影響力)




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Phenothiazines

Metabolic Precursors of the Transmitter Pools of Glutamate and Aspartate, pools of glutamate, aspartate and GABA. Evidence supporting the concept that a-ketoglutarate and malate are supplied by astrocytes to synaptic terminals include the immunohistochemical and biochemical demonstration that pyruvate carboxylase, the principal anaplerotic enzyme in CNS tissues, is locat

Overdose

,Glia–Neuronal Interrelationships in the Metabolism of Excitatory Amino Acids,gh capacity transporter. In addition, glutamine synthesis and catabolism are intimately associated with neurotransmitter glutamate. The enzyme, glutamine synthetase, is highly enriched in astrocytes. The catabolic enzyme, glutaminase, may or may not be enriched in glutamergic nerve endings. Experime

ablate

The Compartmentation and Turnover of Glutamate and GABA: A Better Understanding by the Use of Drugse and GABA can be explained from a two-compartment model. (Berl et al, 1961; Berl and Clarke, 1978; van den Berg and Garfinkel, 1971; Balazs et al, 1970). Such a simple classification is surprising in view of the heterogenous composition of the brain both in anatomical and biochemical terms. The two

BRUNT

Immunocytochemical Localization of Enzymes Involved in the Metabolism of Excitatory Amino Acids,mino acids. Glutaminase is found to be enriched in several putative excitatory amino acid neurons including the auditory nerve, neurons of the hippocampus and cerebral cortex and granule cells of the cerebellum. Cytoplasmic aspartate aminotransferase is enriched in some excitatory amino acid neurons

BRUNT

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Excitatory Amino Acids as Transmitters in the Olfactory System,some olfactory cortical pyramidal cells is reviewed. Several reports describing the synaptically-evoked release of endogenous Asp and/or Glu from olfactory cortex slices support such a role. Surgical and chemical lesions cause loss of Asp and/or Glu at sites corresponding to the location of the cand

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