Titlebook: Etiology of Hodgkin’s Disease; Ruth F. Jarrett Book 1995 Springer Science+Business Media New York 1995 Antigen.Pathogene.epidemiological.e

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,Evidence of an Increased Frequency of HLA-DPB1*0301 in Hodgkin’s Disease Supports an Infectious Aetsis by determining the frequency of HLA-DPB1 alleles in two series of HD patients using molecular typing methods. One series consisted of a retrospective/prospective group of 118 patients over the age of 15 years, and the other a prospective group of 45 patients between the ages of 16 and 24 years.

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Epstein-Barr Virus and Lymphomas: An Overview, EBV infection usually occurs in childhood and, once infected, individuals become life-long virus carriers. The virus is the causative agent of infectious mononucleosis (IM), a self-limiting lymphoproliferative disease resulting from delayed primary EBV infection, and is also associated with a numbe

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,Hodgkin’s Disease and Epstein-Barr Virus, 8 years that a firm molecular basis for this association has been established. The literature relating to EBV and HD has increased exponentially over the latter years and laboratories throughout the world have contributed to a body of data consistently linking this virus with HD. The majority of pa

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,Relationship Between Expression of Cellular Genes and Epstein-Barr Virus in Hodgkin’s Disease,ive” cells. The origin of the neoplastic cells in HD is still controversial (Hsu .., 1985; Cabanillas .., 1988; Hsu ..., 1992; Drexler, 1992). Recent data favour the hypothesis that the RS cells and their mononuclear variants (collectively called H-RS cells) originate from lymphoid cells, although c

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The Reed-Sternberg Cell and the CD30 Antigen,t cells of various types, pathognomonic for HD. In most cases, the H-RS cells account for less than 5% and in some cases for less than 1% of the total tumour mass. In classical cases, the H-RS cells are large cells with bi-lobed or poly-lobed nuclei so that the cells appear binucleated or multinucle

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CD30 Ligand: Cloning, Characterisation and Biological Activities,eed-Sternberg (H-RS) cells in a hyperplastic background of normal, reactive lymphocytes, plasma cells, histiocytes, neutrophils, eosinophils, and stromal cells. The histopathological presentation and characteristic clinical features of HD correlate with an unbalanced production of multiple cytokines