Titlebook: Essentials of Restenosis; For the Intervention Henricus J. Duckers,Elizabeth G. Nabel,Patrick W. Book 2007 Humana Press 2007 Stent.anatomy

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ng. Recent findings have suggested that bone marrow-derived precursors can also give rise to vascular cells that contribute to repair, remodeling, and lesion formation. This article overviews recent findings on circulating vascular progenitor cells and describes potential therapeutic strategies that target these cells.

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The Genomics of Restenosisw technique involving stent deployment had been designed to overcome vessel recoil (.), initially, considered the major contributor to the loss of lumen diameter. Rather than ridding interventional cardiology of restenosis, a purely iatrogenic process, stents have shifted the focus of attention toward the phenomenon of intimal hyperplasia (.).

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Contribution of Circulating Progenitor Cells to Vascular Repair and Lesion Formationng. Recent findings have suggested that bone marrow-derived precursors can also give rise to vascular cells that contribute to repair, remodeling, and lesion formation. This article overviews recent findings on circulating vascular progenitor cells and describes potential therapeutic strategies that target these cells.

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Arterial Remodelingsease (.–.). Expansive remodeling prevents and constrictive remodeling accelerates narrowing of the lumen (.–.). Also in restenosis after balloon angioplasty, in both experimental research . as well as in humans (.), constrictive remodeling is the most important determinant of luminal renarrowing (.).

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Book 2007ts, and other pioneers in the field cover everything from non-invasive imaging, to eluting stents, to brachytherapy through to the latest molecular biology-based treatments including antisense, stem cells and gene therapy, making this an essential reference for all interventional cardiologists.

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https://doi.org/10.1057/9781137009524relevance for cardiovascular disease. These include functional subsets of macrophages and T-lymphocytes, newly identified receptors for pathogens, the reciprocal regulation of lipid metabolism, inflammation and infection, and the hygiene hypothesis.

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