Titlebook: Essential Hypertension 2; Kyuzo Aoki Book 1989 Springer Japan 1989 hypertension

IRK

Excitatory Agonists and Ca-Permeable Channels in Arterial Smooth Muscle Cellsd. Stimulation with norepinephrine is not accompanied by a depolarization of the cell membrane. The opening of voltage-gated Ca channels by depolarization can therefore be excluded. The existence of receptor-operated Ca channels, which are activated by a direct interaction of the agonist with its re

飛鏢

松雞

輕而薄

Myogenic Contraction and Relaxation of Arterial Smooth Muscleproduced by stretch, agonistic stimuli, and high K. in helical strips of canine cerebral artery. Simultaneous measurements of the Ca. signals and tension development were made with a fluorimeter. The autofluorescence, motion artifact, and Ca. buffering action, considered as main disadvantages when f

RLS898

Relationship Between Cytosolic Calcium Level and Contractile Tension in Vascular Smooth Muscle). High-K., norepinephrine, prostaglandin F., and ionomycin induced sustained contraction following sustained increase in [Ca.]., whereas Bay k 8644 and tetraethylammonium caused rhythmic contractions following the rhythmic changes in [Ca.].. Tension changes were always preceded by the changes in [C

freight

僵硬

Sarcoplasmic Reticulum of Mesenteric Arteries Buffers Stimulated Ca2+ Entryall mesenteric arteries to Ca.-free experimental solutions containing EGTA with or without agonist. The agonist-mediated Ca. depletion of the SR markedly reduced the rate and magnitude of the contraction due to Ca. influx through voltage-gated channels. This buffering of Ca. influx by the norepineph

正式演說

出汗

MELD