Titlebook: Essential Hypertension; Calcium Mechanisms a Kyuzo Aoki Book 1986 Springer-Verlag Tokyo 1986 hypertension.treatment

人工合成

書目名稱Essential Hypertension影響因子(影響力)




書目名稱Essential Hypertension影響因子(影響力)學(xué)科排名




書目名稱Essential Hypertension網(wǎng)絡(luò)公開度




書目名稱Essential Hypertension網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Essential Hypertension被引頻次




書目名稱Essential Hypertension被引頻次學(xué)科排名




書目名稱Essential Hypertension年度引用




書目名稱Essential Hypertension年度引用學(xué)科排名




書目名稱Essential Hypertension讀者反饋




書目名稱Essential Hypertension讀者反饋學(xué)科排名

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爆米花

克制

Actions of Calcium Agonists and Antagonists on Femoral Arteries of Spontaneously Hypertensive Ratsthe data compared with findings in normotensive Wistar-Kyoto rats (WKY). The addition of Bay k 8644 produced a dose-dependent contraction in the SHR femoral artery with a pD. value of 8.55. The maximum contraction induced by this Ca. agonist (1 x 10–..) was comparable with the maximum developed by e

實現(xiàn)

Altered Vascular Calcium Metabolism As a Possible Cause of Increased Blood Pressure in Essential Hyphere is little information from human studies as to whether such abnormalities play a role in the development of hypertension, but from rat studies the available evidence does not support a direct connection between EC coupling abnormalities and increased blood pressure, although the possibility can

Nefarious

Calcium-Membrane Interactions in Smooth Muscle in Relation to Hypertensionractionation technique with special reference to the relationship between the alterations of calcium ion handling in vascular smooth muscle and the pathogenesis of spontaneous hypertension in rats. The calcium-membrane interactions include ATP-dependent Ca-transport, Na-dependent Ca-transport (Na-Ca

Nefarious

peritonitis

我要威脅

Growth Factors and Vascular Smooth Muscle Cells in Spontaneously Hypertensive Ratsth increased synthesis of collagen and noncollagen protein. Cultured VSMCs from rat aorta appear to be a suitable in vitro system to study the mechanism of cellular proliferation of VSMC, because they are completely free from mechanical and neurohumoral influence. Therefore, the present study was de

FOIL

Calcium and Calcium Antagonists on Vascular Damage in Spontaneously and Dahl Hypertensive Rats of transmembrane calcium influx by nifedipine prevented the genetically determined hypertension, cardiac hypertrophy, and renal ischaemia in Aoki spontaneously hypertensive rats. In salt-sensitive Dahl rats, nifedipine and its calcium antagonistic dihydropyridine derivatives prevented vascular fibr