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Titlebook: Esophageal Carcinoma; State of the Art Jochen Lange,J. R. Siewert Conference proceedings 2000 Springer-Verlag Berlin Heidelberg 2000 Stagin

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41#
發(fā)表于 2025-3-28 15:36:22 | 只看該作者
https://doi.org/10.1007/978-981-15-8225-7lantation. The patient’s voice may be preserved by means of tracheopharyngeal shunts with intestinal interposition. The advances of radiation therapy and chemotherapy will enable less extended resections with greater rates of laryngeal preservation.
42#
發(fā)表于 2025-3-28 19:24:27 | 只看該作者
Climate Change Policies in Australia,liative treatment of obstructing tumors but precancerous lesions such as Barrett’s esophagus with or without dysplasia and early cancer. PDT might establish itself as a minimally invasive treatment alternative compared with surgery for high-grade dysplasia or early mucosal cancer of the esophagus.
43#
發(fā)表于 2025-3-28 22:53:56 | 只看該作者
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發(fā)表于 2025-3-29 07:05:11 | 只看該作者
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發(fā)表于 2025-3-29 10:33:21 | 只看該作者
Therapy of Cervical Esophageal Carcinoma,lantation. The patient’s voice may be preserved by means of tracheopharyngeal shunts with intestinal interposition. The advances of radiation therapy and chemotherapy will enable less extended resections with greater rates of laryngeal preservation.
46#
發(fā)表于 2025-3-29 13:52:16 | 只看該作者
47#
發(fā)表于 2025-3-29 19:05:00 | 只看該作者
48#
發(fā)表于 2025-3-29 22:03:01 | 只看該作者
Modern Pathology: Prognostic Parameters in Squamous Cell Carcinoma of the Esophagus,n of the proliferation regulating molecule p 21. and weak expression of the apotosis regulating molecule Bcl-X. were predictors of poor survival in univariate and multivariate survival analysis. No prognostic impact was shown in relation to the expression of p 53 and the apoptosis regulating molecul
49#
發(fā)表于 2025-3-30 01:16:05 | 只看該作者
50#
發(fā)表于 2025-3-30 06:27:16 | 只看該作者
,Malignant Degeneration of Barrett’s Esophagus: Clinical Point of View, of Barrett’s esophagus, although appealing, currently constitutes a potentially dangerous procedure without proven benefit for the patient. Since the development of Barrett’s adenocarcinoma follows a multistep process from metaplasia through increasingly severe grades of dysplasia, close endoscopic
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