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Titlebook: Erythropoietin and the Nervous System; Ahmet H?ke (Associate Professor of Neurology and N Book 2006 Springer-Verlag US 2006 Nervous System

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發(fā)表于 2025-3-23 09:55:13 | 只看該作者
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發(fā)表于 2025-3-23 15:07:01 | 只看該作者
International Management Knowledgepanied by an inflammatory reaction including production of inflammatory cytokines, leukocyte infiltration, and astroglial activation/ proliferation. In models of middle cerebral artery occlusion, EPO decreases production of tumor necrosis factor (TNF), interleukin (IL)-6, and of monocyte chemoattrac
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發(fā)表于 2025-3-23 22:01:20 | 只看該作者
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發(fā)表于 2025-3-23 23:16:10 | 只看該作者
978-1-4899-7383-2Springer-Verlag US 2006
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發(fā)表于 2025-3-24 03:32:07 | 只看該作者
https://doi.org/10.1007/978-0-387-30011-5Nervous System; brain; central nervous system; death; hypoxia; inflammation; neuroprotection; neuroscience;
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發(fā)表于 2025-3-24 06:52:06 | 只看該作者
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發(fā)表于 2025-3-24 12:15:40 | 只看該作者
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發(fā)表于 2025-3-24 18:44:34 | 只看該作者
Erythropoietin in Spinal Cord Injury,ic, traumatic and inflammatory SCI. The recent development of non-erythropoietic derivatives of EPO with outstanding preclinical characteristics encourages evaluation of tissue-protective cytokines in clinical trials of spinal cord injury.
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發(fā)表于 2025-3-24 22:43:47 | 只看該作者
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發(fā)表于 2025-3-25 01:59:54 | 只看該作者
as to generate carbamylated EPO (CEPO), which has no affinity for the EPO receptor, but the same neuroprotective potency as EPO. Our data suggest that the cytoprotective signal transduction of EPO is distinct from the hematopoietic signaling machinery.
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