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Titlebook: Epstein Barr Virus Volume 1; One Herpes Virus: Ma Christian Münz Book 2015 The Editor(s) (if applicable) and The Author(s), under exclusive

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發(fā)表于 2025-3-21 17:12:48 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Epstein Barr Virus Volume 1
副標(biāo)題One Herpes Virus: Ma
編輯Christian Münz
視頻videohttp://file.papertrans.cn/314/313401/313401.mp4
概述Epstein on the discovery of EBV 50 years ago.32 comprehensive chapters on EBV virology, tumorigenesis and immune control.Newest insights on EBV strain variation, the recombinant EBV system and in vivo
叢書(shū)名稱Current Topics in Microbiology and Immunology
圖書(shū)封面Titlebook: Epstein Barr Virus Volume 1; One Herpes Virus: Ma Christian Münz Book 2015 The Editor(s) (if applicable) and The Author(s), under exclusive
描述Epstein Barr virus (EBV) was discovered as the first human tumor virus around 50 years ago.?Since its discovery in Burkitt’s lymphoma it has been associated with various other malignancies, infectious mononucleosis and even autoimmune diseases. The two book volumes on EBV summarize the first 50 years of research on this tumor virus, starting with historical perspectives on discovery, oncogenicity and immune control, reviewing the role that the virus plays in the various associated diseases and concluding with a discussion on how the immune system keeps persistent EBV infection under control in healthy EBV carriers and can be used to treat EBV associated diseases. The respective 32 chapters are written by international experts from three continents for health care providers, biomedical researchers and patients that are affected by EBV. The assembled knowledge should help to understand EBV associated diseases better and to develop EBV specific vaccination in the near future.
出版日期Book 2015
關(guān)鍵詞cell-mediated immune control; human tumor virus; immunotherapies; lymphomas and carcinomas; persistent i
版次1
doihttps://doi.org/10.1007/978-3-319-22822-8
isbn_softcover978-3-319-37177-1
isbn_ebook978-3-319-22822-8Series ISSN 0070-217X Series E-ISSN 2196-9965
issn_series 0070-217X
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerl
The information of publication is updating

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https://doi.org/10.1007/978-3-642-91308-2ions contribute to the regulation of viral gene expression, DNA replication, and episome persistence during latency. This review focuses on how EBV latency is regulated by chromatin and its associated processes.
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Anleitung zur Untersuchung der Lebensmitteln and the etiology of endemic BL, the modulation of viral persistence in children by malaria and deficits in EBV immunosurveillance has more recently been reified. Renewed efforts to design prophylactic and therapeutic EBV vaccines provide hope of preventing EBV-associated BL as well as increasing the ability to cure this cancer.
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Bestimmung der Aethylimidgruppe ,]], for systemic lupus erythematosus (SLE), whereas evidence is either lacking or not supportive for other autoimmune conditions. In this chapter, we present the main epidemiological findings that justify the conclusion that EBV is a component cause of MS and SLE and possible mechanisms underlying these effects.
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Book 2015ciated with various other malignancies, infectious mononucleosis and even autoimmune diseases. The two book volumes on EBV summarize the first 50 years of research on this tumor virus, starting with historical perspectives on discovery, oncogenicity and immune control, reviewing the role that the vi
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