Titlebook: Epitope Recognition Since Landsteiner’s Discovery; 100 Years Since the Martha Eibl,W. R. Mayr,G. J. Thorbecke Conference proceedings 2002

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Antigen Recognition: 100 Years After Landsteiner,the past century to understand this extraordinary ability grew largely from Landsteiner’s work on the reactions of antisera with the small organic molecules he called haptens, or partial antigens, to indicate that they could react specifically with antibodies but not elicit antibody production. It w

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Using Monoclonal Antibodies and Site Directed Mutagenesis to Map the Epitopes of the Blood Group Rhen her husband’s blood. Her serum agglutinated her husband’s red cells and those of 80% of ABO compatible donors. They showed that this new antigen, to which they did not assign a name, was different to the then known blood groups ABO, MN and P. They hypothesised that the mother had been immunised t

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Infections and the Immune Response to Cardiac Antigens,of morbidity and mortality in rich countries. In recent years, infections with various pathogens have emerged as risk factors for cardiovascular disease. While a causal dependence has not yet been established, experimental animal models were useful to gain insights into potential mechanisms linking

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VLSI process engineering operations, after single intravenous injections of only 50μg of the PLP139-151 16-mer, administered to SJL/J mice 7 d after the induction of the disease. Although relapse occurred at about day 45, an additional injection several days before that maintained the suppression. Importantly, the specific suppressive

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Antigen Recognition: 100 Years After Landsteiner,al monograph on . he cautiously attributed the specificity of antisera to a “….” (1). By agents, he meant antibodies, of course, and by substrata he meant antigens or haptens. Today, having the benefit of over 60 years of further intensive study in many laboratories we can define specificity simply

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Induction and Suppression of an Autoimmune Disease by Oligomerized T Cell Epitopes: Enhanced In Viv after single intravenous injections of only 50μg of the PLP139-151 16-mer, administered to SJL/J mice 7 d after the induction of the disease. Although relapse occurred at about day 45, an additional injection several days before that maintained the suppression. Importantly, the specific suppressive