Titlebook: Epithelial—Mesenchymal Interactions in Cancer; I. D. Goldberg,E. M. Rosen Book 1995 Birkh?user Verlag 1995 Activation.Grading.biology.canc

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Robert M. Zacharko,Hymie Anismancogene product Met, including mitogenicity, motility, and morphogenesis. Normally, . expression is restricted to epithelial cells and is activated in a paracrine fashion by HGF/SF secreted from cells of mesenchymal origin. In this chapter, we review data showing that: (i) . over-expression in HGF/SF

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The Glucose Tolerance Test in Mice, sites. Cancer metastases form by way of a complex series of sequential steps involving a variety of tumor cell and host properties. These steps typically involve growth and invasion of malignant cells at primary sites, followed by their penetration into lymphatics and blood circulation and body cav

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Acquisition of Drug Self-Administrationile fashion to a variety of external ligands including autocrine motility factors, growth factors, and components of the extracellular matrix. Since tumor cell motility is a necessary component of tumor invasion and metastasis, we speculate that these protein factors could play important regulatory

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Animal Models of Drug Addiction,ll destruction at the site of ovulation, (2) epithelial and stromal cell proliferation and migration into the ruptured follicle, and (3) remodeling and re-epithelialization of the folicular ‘wound’ (Nicosia et al., 1989; Dietl and Marzusch, 1993). These changes in ovarian epithelial and stromal (fib

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