Titlebook: Epidemiology of Alzheimer’s Disease: From Gene to Prevention; Richard Mayeux,Y. Christen Conference proceedings 1999 Springer-Verlag Berli

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Dmitry S. Yershov,Steven M. LaValle the predominant, late-onset form (90–95% of the patients). The APOE4 allele may explain up to 17% of AD in the general population. A key issue to resolve from a public health point of view will be the interaction of this gene with drugs and other genetic and non-genetic risk factors. Large scale, l

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Springer Tracts in Advanced Roboticse disease. Ultimately, to understand and to defeat the disease, we must understand the new cohorts of people with the disease, their genetic, environmental and social characteristics and the interactions of those characteristics. Furthermore, if we are to enlist participants in research and develop

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Algorithmic Foundations of Robotics VIa dose-response relationship has been observed, such that women who have greater estrogen exposures (higher doses or longer durations of therapy) also have lower risks of developing Alzheimer’s disease. An association between Alzheimer’s disease and estrogen has not been discerned in all studies, ho

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Jeffrey Ichnowski,Ron Alterovitz995). In the Baltimore Longitudinal Study of Aging (Stewart et al. 1997), prospective data suggested that use of NSAIDs for more than two years may reduce the risk of AD by approximately one half. Many studies have been identified by McGeer et al. (1996) as suggesting a protective effect of anti-inf

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Data Science, Machine Intelligence, and Lawth et al. 1995). These costs ignore the enormous impact, both social and economic, on care givers. Dementia, therefore, represents a major public health problem for Western society and one that is only likely to increase over the next several decades, given the current demographic trends. The impact

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Epidemiology of Alzheimer’s Disease: From Gene to Prevention

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