Titlebook: Energy Metabolism and Obesity; Research and Clinica Patricia A. Donohoue Book 2008 Humana Press 2008 Balance.Diabetes.Energy.Insulin.Insuli

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The Efferent Arm of the Energy Balance Regulatory Pathway: Neuroendocrinology and Pathology,rough afferent neural and hormonal signals reaching the hypothalamus, with resultant efferent projections of the autonomic nervous system innervating the muscles, viscera, and adipose tissue. The efferent system is of particular interest, as it has its own pathologies, which can be modulated to achi

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Interaction Between Physical Activity and Genetic Factors in Complex Metabolic Disease,essed carbohydrates is likely to have increased and physical activity levels declined. However, the rates at which obesity and diabetes have increased differ across people of varying ethnicities living in the same environment, suggesting the presence of interaction between ethnic-specific factors, s

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,Antipsychotic Medication–Induced Weight Gain and Risk for Diabetes and Cardiovascular Disease, and cardiovascular disease (CVD), with related increases in mortality. Increased adiposity is associated with decreases in insulin sensitivity, leading to increased risk of hyperglycemia and hyperlipidemia. Current evidence supports the hypothesis that treatment with antipsychotic medications is as

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The Surgical Approach to Morbid Obesity,inning in 1966, which restricts food intake and produces less malabsorption. Both operations exposed the distal ileum to glucose and fat, which stimulates secretion of glucagon-like peptide-1 (GLP-1). This is a brake hormone for gastric emptying and intestinal peristalsis. GLP-1 is also an incretin

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Zulejha Merhemic,Majda M. Thurnhertaining normal body weight in humans is highlighted by the finding that disordered melanocortin signaling results in early-onset morbid obesity and dramatic increases in lean body mass in humans. Emerging evidence suggests that blocking this system via pharmacologic antagonists to the type 4 melanoc