Titlebook: Eicosanoids in Inflammatory Conditions of the Lung, Skin and Joints; Martin Church,Clive Robinson Book 1988 MTP Press Limited 1988 asthma.

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Eicosanoids in Immune Regulation,h mediate their effects. Finally, we are still in the process of dissecting the cellular and molecular nature of immune responses; thus, information derived from experiments conducted with LO products and inhibitors must be evaluated with the understanding that the mechanism of any effect may be unc

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Current Concepts on the Role of Eicosanoids in the Pathogenesis of Bronchial Asthma,untered in the clinic, i.e. where sensitization has already taken place, is set against an established background of bronchial hyper-reactivity. This is also true of the subjects who participate in studies designed to investigate the cellular and molecular basis of asthma. It is likely that the bron

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K. Seshagirirao,M. N. V. Prasadhapter, the action of a given mediator alone is often insufficient to induce an overt inflammatory response and is dependent on an interaction, or synergism, with another mediator to produce the features described above.

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predictive value, is open to question. With this point in mind, in this chapter, we will consider the relevance of a variety of experimental models of inflammation in which the contribution of eicosanoids has been investigated. We have chosen, as examples, animal models of asthma, other pulmonary diseases and rheumatoid arthritis.

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The Cellular and Molecular Basis of Eicosanoid Generation, which have a plethora of biological functions. The possibility that these products, which include prostanoids and leukotrienes, are intimately related with the pathogenesis of inflammatory and allergic diseases has fascinated scientists and clinicians alike since long before their chemical characterization.