Titlebook: Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation, and Radiation Injury 2; Part A Kenneth V. Honn,Santosh Nigam,Lawrence J. Ma

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Eric C. K. Cheng,Joanna K. M. Chantood. Lipid peroxidation, phospholipase A. activity and myeloperoxidase activity were increased in colorectal cancers compared with macroscopically normal tissues (Otamiri et al. 1989). Varying amounts of total arachidonic acid in the phospholipids of colorectal tumors compared to normal associated

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https://doi.org/10.1057/9780230299016 which contains a non-heme iron. The common characteristics of the reaction catalyzed by .-type lipoxygenases (LOX) include:. i) substrates with a .-1,4-pentadiene structure, preferably in afree fatty acid, ii) a three step reaction mechanism consisting of hydrogen abstraction at C(3) of the pentadi

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https://doi.org/10.1007/978-981-15-3738-7(2–4), as well as in human (5,6) and rodent tumor cells (7,8). In tumor cells, the major metabolite, 12(S)-HETE, regulates their metastatic potential (9–11) through a protein kinase C-dependent mechanism (7). The intracellular distribution of 12-LOX activity varies between different cells ranging fr

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Ying Xu,Glenn Patmore,Paul J Gollan is the first step in prostaglandin synthesis, and is also the rate limiting step in this pathway. In addition, the PHS protein is inactivated at a rapid rate during catalysis. Thus, the regulation of PHS expression has a direct and significant effect on the rate of prostaglandin synthesis.

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https://doi.org/10.1007/978-0-387-71485-1eicosanoids. Eicosanoid formation has been shown to be elicited by a number of particulate (bacteria, viruses, zymosan, immune complexes) and soluble (phorbol ester, PAF, C3a, fluoride, calcium ionophore, LPS, cytokines) agents. The release of arachidonic acid (AA) and eicosanoids in these cells has