Titlebook: Effects of Mineral Dusts on Cells; Brooke T. Mossman,Raymond O. Bégin Conference proceedings 1989 Springer-Verlag Berlin Heidelberg 1989 A

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Imbalanced Data and Resampling Techniques,mmation and, in the longer term, septal fibrosis and epithelial abnormalities (Begin .. 1986; Gibbs .. 1984). We have been studying the role of inflammatory leukocytes in the development of pathology in dust-exposed lung and have previously reported on the ability of inflammatory bronchoalveolar leu

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Data Monitoring Committees (DMCs)million tons of palygorskite was used in industrial countries of the western world, as compared to 400,000 tons of sepiolite in 1984 (see IARC, 1987, for production, consumption, and application data). Concern about the potential health hazard of these minerals has arisen due to their fibrous nature

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Susan S. Ellenberg,Jay P. Siegel al,1985), despite the distinctly different potentials of quartz and clay for causing pneumoconiosis or pulmonary fibrosis (Sheers, 1984). A respired particle depositing in a respiratory bronchiole or alveolus will contact a pulmonary surfactant hypophase, of which a primary component is diacyl glyc

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Communication and Synchronization,Coal worker’s pneumoconiosis is usually divided in two groups: simple pneumoconiosis (SP) and progressive massive fibrosis (PMF). This distinction is of importance in clinical practice since PMF is definitively disabling. However, why some workers should develop PMF when exposed to dust concentratio

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https://doi.org/10.1007/978-1-4842-3967-4 in turn influenced by .. dissolution. This study was conducted to investigate the role of differing physical and chemical properties of beryllium and nickel compounds on their cytotoxicity to cell systems ... We hypothesized that the two major factors influencing cytotoxicity are chemical form and

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Robert Walters,Leon Trakman,Bruno Zellerular zymogen plasminogen into active plasmin, another neutral protease. PA is thought to play an important role in regulating proteolytic events that occur in various normal conditions, but also in pathological processes involving tissue destruction and cell migration as, for instance, inflammatory

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Data Protection in a Profiled Worldacrophage-Iike cell line (J774A.1) in vitro. Following 24- and 48-hour incubations, the cells were observed under phase contrast microscopy for changes in cell morphology and cell viability was assessed using trypan blue exclusion. A viability index (VI) was calculated to reflect changes in both cel