Titlebook: Early Indicators Early Treatments Neuroprotection in Multiple Sclerosis; Otto R. Hommes,Giancarlo Comi Book 2004 Springer-Verlag Italia 20

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Genetic Regulation of Nerve Cell Death/Glial Activation and Protective Effects of Myelin Basic Proten 5% and 60% [7, 10, 11]. Notably, no single HLA type seems to exclude MS. It is therefore clear that as yet unidentified genes residing outside the HLA must also be of importance for disease susceptibility in MS.

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Monounsaturated Fatty Acids and Neuroprotection. The Results of a Study of Cognitive Decline in Oldmore-detailed procedure for the assessment of cognitive decline than those listed in the AAMI criteria. A high incidence (45%) of dementia was found in individuals aged >75 years who were diagnosed as having “minimal dementia” by the CAMDEX interview [7]. Furthermore, in another study 48% of patient

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Early Treatment in Multiple Sclerosis with Intravenous Immunoglobulin: Rationale and Study Design,ctive of this study was to investigate the efficacy of IVIg treatment, administered at 0.4 g/kg per day for 5 consecutive days, as a loading dose, and 0.4 g/kg per day every 6 weeks thereafter as a booster dose, for a 1-year period, on the risk of patients with clinically probable MS and positive br

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The Yin and Yang of Inflammation in Multiple Sclerosis,neity occurs is currently unknown. Are RR and PPMS different diseases or are they part of the same clinical spectrum? Why do some patients develop progressive disease whilst others do not? Answers to these questions will not only improve our understanding of MS, but will also have major implications

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Book 2004fect. Pathological studies in multiple sclerosis have now clearly demonstrated that destructive processes may be followed by recovery phases in such a way that myelin may be morphologically and functionally reconstituted. These findings provide the rationale for early treatment and emphasize the imp

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