Titlebook: Early Arrhythmias Resulting from Myocardial Ischaemia; Mechanisms and Preve James R. Parratt Book 1982 The contributors 1982 arrhythmia.car

冰河期

chemicals like aconitine, chloroform or ouabain. If promising results are obtained in these tests then the potential antiarrhythmic drug may be tested (usually in conscious dogs) for its ability to suppress ventricular ectopic activity ensuing 1–2 days after the production of myocardial ischaemia.

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滑稽

ythmia are unclear. Complete coronary occlusion is frequently absent in victims of sudden death (Basche ., 1975) and experimental studies indicate that both coronary occlusion and reperfusion are associated with ventricular fibrillation (Penkoske ., 1978). The picture is further complicated by the f

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Credence

https://doi.org/10.1007/978-94-011-0733-4 1962 a study of arteriosclerotic patients by Murphy and Mustard revealed, by chance, that those taking sulphinpyrazone had prolonged platelet survival and reduced platelet turnover; this effect was confirmed by Smythe . (1965), who noted also reduced platelet adhesiveness. These findings were demon

空洞

,Sudden Cardiac Death — An Overview,nt medical advance (Zoll ., 1956), because the brevity of the event pre-empted studies of its causes (and still does) and because it commonly occurs without praecordial tightness or any precise clinical syndrome.

ALIAS

Myocardial Ischaemia and Metabolic Changes Associated with the Genesis of Ventricular Arrhythmias,ischaemia. The objective of this chapter is to identify the ischaemia-induced changes which may be critical to the genesis of arrhythmias. In order to achieve this it will be necessary to consider the nature and progression of ischaemic injury.

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conception