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Titlebook: E. coli; Shiga Toxin Methods Dana Philpott,Frank Ebel Book 2003 Humana Press 2003

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發(fā)表于 2025-3-21 17:53:49 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱E. coli
副標題Shiga Toxin Methods
編輯Dana Philpott,Frank Ebel
視頻videohttp://file.papertrans.cn/301/300216/300216.mp4
概述Includes supplementary material:
叢書名稱Methods in Molecular Medicine
圖書封面Titlebook: E. coli; Shiga Toxin Methods  Dana Philpott,Frank Ebel Book 2003 Humana Press 2003
描述The study of the pathogenesis of Shiga toxin-producing Escherichia coli (STEC) infections encompasses many different disciplines, including clinical microbiology, diagnostics, animal ecology, and food safety, as well as the cellular microbiology of both bacterial pathogenesis and the mechanisms of toxin action. E. coli: Shiga Toxin Methods and Protocols aims to bring together a number of experts from each of these varied fields in order to o- line some of the basic protocols for the diagnosis and study of STEC pat- genesis. We hope that our book will prove a valuable resource for the clinical microbiologist as well as the cellular microbiologist. For the clinical microbiologist, our aim is to detail a number of current protocols for the detection of STEC in patient samples, each of which have their own advantages. Chapter 1 provides an introduction into the medical significance of STEC infections. Chapters 2–7 follow with protocols for the diagnosis and detection of STEC bacteria in patient and animal samples.
出版日期Book 2003
版次1
doihttps://doi.org/10.1385/159259316X
isbn_softcover978-1-61737-264-3
isbn_ebook978-1-59259-316-3Series ISSN 1543-1894 Series E-ISSN 1940-6037
issn_series 1543-1894
copyrightHumana Press 2003
The information of publication is updating

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沙發(fā)
發(fā)表于 2025-3-21 21:53:22 | 只看該作者
STEC in the Food Chain,y all of STEC strains contain the . attaching and effacing gene (.) encoding the outer membrane protein intimin, which mediates the attachment to enterocytes leading to irreversible destruction of the microvilli (.).
板凳
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地板
發(fā)表于 2025-3-22 07:13:55 | 只看該作者
Rong Xu,Richard J. Simpson,David W. Greeningc discipline “cellular microbiology” focuses on the moleular crosstalk between pathogens and their host cells, as the elucidation of these interactions not only helps to understand the molecular basis of disease but has also given rise to new model systems for probing host cell functions.
5#
發(fā)表于 2025-3-22 09:31:06 | 只看該作者
Baryonium - Nuclear Atom or Colour Molecule?rriers for cellular immunization (.–.), and as an antineoplastic agent (.–.). These various fields of study require the availability of purified toxin and/or its subunits. In order to facilitate the expansion of such studies, in the absence of a commercial supplier, a detailed purification scheme for Stx1 is presented in this chapter.
6#
發(fā)表于 2025-3-22 15:46:08 | 只看該作者
Nuclear Models for High Spin Phenomena,nellar membranes also lose integrity and are unable to maintain normal function. Condensation of chromatin may occur, but it tends to be irregular. Leakage of cytoplasmic contents evokes an inflammatory response in the surrounding tissues. Overall, the configuration of the necrotic cell is maintained until its removal by professional phagocytes.
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發(fā)表于 2025-3-23 04:58:03 | 只看該作者
Methods for the Purification of Shiga Toxin 1,rriers for cellular immunization (.–.), and as an antineoplastic agent (.–.). These various fields of study require the availability of purified toxin and/or its subunits. In order to facilitate the expansion of such studies, in the absence of a commercial supplier, a detailed purification scheme for Stx1 is presented in this chapter.
10#
發(fā)表于 2025-3-23 05:51:08 | 只看該作者
Detection of Shiga Toxin-Mediated Programmed Cell Death and Delineation of Death-Signaling Pathwaysnellar membranes also lose integrity and are unable to maintain normal function. Condensation of chromatin may occur, but it tends to be irregular. Leakage of cytoplasmic contents evokes an inflammatory response in the surrounding tissues. Overall, the configuration of the necrotic cell is maintained until its removal by professional phagocytes.
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