Titlebook: Dyslipidemias in Kidney Disease; Adrian Covic,Mehmet Kanbay,Edgar V. Lerma Book 2014 Springer Science+Business Media New York 2014 Dialysi

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Actin Regulation and Surface Catalysisensity lipoprotein cholesterol are normal or low. To further complicate things, pathologic findings of arterial lesions in CKD consist of calcium-rich atherosclerotic plaques, whereas in classic atherosclerotic disease lipid-laden atheromatous or fibroatheromatous plaques are detected, implying a di

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Cholecystokinin

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https://doi.org/10.1007/978-3-322-91381-4compared with their HD counterparts. Modifications in some parameters of HD or PD along with pharmacologic therapy, mainly statins, constitute the current therapeutic armamentarium against dyslipidemia in dialysis patients. Future randomized controlled trials taking into account the particular chara

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Phenomenology and Its Implications,ategories of pediatric patients are discussed in light of the limited number of studies in this population. Concerning the differences between the disease profile of adults and children, adult data could not be directly translated in children. Thus, prospective investigations regarding the clinical

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How Lipid-Lowering Agents Work: The Good, the Bad, and the Ugly,sis patients. Although fibrates can be used to treat mixed dyslipidemia, they need to be used carefully in patients with CKD, and limited available data suggest that fibrates may have a place in reducing cardiovascular risk in patients with mild to moderate CKD.

流眼淚

CVD in CKD: Focus on the Dyslipidemia Problem,ensity lipoprotein cholesterol are normal or low. To further complicate things, pathologic findings of arterial lesions in CKD consist of calcium-rich atherosclerotic plaques, whereas in classic atherosclerotic disease lipid-laden atheromatous or fibroatheromatous plaques are detected, implying a di