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Titlebook: Dynamics of Brain Edema; Proceedings of the 3 Hanna M. Pappius (Associate Professor, Associate N Conference proceedings 1976 Springer-Verla

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書(shū)目名稱Dynamics of Brain Edema
副標(biāo)題Proceedings of the 3
編輯Hanna M. Pappius (Associate Professor, Associate N
視頻videohttp://file.papertrans.cn/285/284013/284013.mp4
圖書(shū)封面Titlebook: Dynamics of Brain Edema; Proceedings of the 3 Hanna M. Pappius (Associate Professor, Associate N Conference proceedings 1976 Springer-Verla
描述A workshop on Dynamic Aspects of Cerebral Edema was organized to pro- vide an opport~nitY,for interdisciplinary and detailed consideration of this subject, so crucial in neurology and neurosurgery. The previ- ous workshops were held in Vienna in 1965 and in Mainz in 1972. In the meantime, our ideas on mechanisms of resolution of cerebral edema had been changing drastically. Controversy had arisen regarding the role of biogenic amines in the development of edema. Active work in several centers had indicated the possibility of a reversible compo- nent in edematous changes associated with ischemia, the control of which could be of therapeutic significance in the problem of stroke. It was felt that a thorough discussion, at this time, by those involv- ed in these various studies should help to resolve the controversies, to crystallize the implications of the new findings, and to evaluate their application for patient care. In preparing the proceedings for publication we have edited the manu- scripts and selected the discussions with an aim to highlight new find- ings, to make this volume readable, and to eliminate duplication. We gratefully acknowledge financial support from the instit
出版日期Conference proceedings 1976
關(guān)鍵詞brain; cerebral ischemia; head; head injury; histology; intracranial pressure; neurology; neurosurgery; spin
版次1
doihttps://doi.org/10.1007/978-3-642-66524-0
isbn_softcover978-3-540-08009-1
isbn_ebook978-3-642-66524-0
copyrightSpringer-Verlag Berlin · Heidelberg 1976
The information of publication is updating

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Pursuing Practice Wisdom in Leadershiptic dehydrogenase and total protein, and clinical signs of increased intracranial pressure. With resolution of CSF pressure, there is a return to baseline of CSF chemistry and partial resolution of the other parameters. The cerebral blood flow shows a greater recovery in gray than white matter, but
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Pursuing Practice Wisdom in Leadershipf platelets in the small cerebral vessels, associated with preservation of the abnormal arterial circulation, may be an important factor in the pathogenesis of late wide-spreading brain edema after cryogenic injury to the cortex.
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Central Noradrenergic Regulation of Brain Microcirculation in water permeability and a decrease in cerebral blood flow was also induced by intracarotid infusion of hypertonic urea. Neurally mediated mechanism for regulation of brain water permeability is proposed which would account for changes in permeability induced by electrical stimulation and by mecha
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Determination of Cerebral Edema by Quantitative Morphometryce the volume) of astrocytic foot processes were significantly greater in the permeable areas of the hypertensive animals than in the adjacent nonpermeable areas from the same animals and from comparable areas in normotensive animals..It was concluded that the edema was confined to the permeable are
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Delayed Vasogenic Edema Following Irradiation of the Monkey Braintic dehydrogenase and total protein, and clinical signs of increased intracranial pressure. With resolution of CSF pressure, there is a return to baseline of CSF chemistry and partial resolution of the other parameters. The cerebral blood flow shows a greater recovery in gray than white matter, but
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Early Hemodynamic Changes at the Microcirculatory Level Following Focal Cryogenic Injury over the Cof platelets in the small cerebral vessels, associated with preservation of the abnormal arterial circulation, may be an important factor in the pathogenesis of late wide-spreading brain edema after cryogenic injury to the cortex.
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Triethyl Tin-Induced Cerebral Edema: Implications for Determination of Cerebral Blood Flow in Edemat
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