Titlebook: Drugs for the Treatment of Parkinson’s Disease; Donald B. Calne (Head) Book 1989 Springer-Verlag Berlin Heidelberg 1989 Parkinson.adverse

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Neurophysiology of Basal Ganglia, deficits in the basal ganglia would suggest at first sight a role of this brain structure in motor control. However, an absence of movement can have many causes. When discussing movement-related neuronal processes it is helpful to distinguish between the initiation and the execution of movements (.

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Biochemical Neuroanatomy of the Basal Ganglia,opic since the early days of biochemical neuroanatomy because many of the classical neurotransmitters are found in very high concentration in extrapyramidal nuclei, these nuclei are relatively distinct and easily dissected, and classical pathological studies had demonstrated a clear association betw

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Receptors in the Basal Ganglia, the function of specific neuronal pathways in health and disease. In Parkinson’s disease, the emphasis has been on the study of dopamine presynaptic and postsynaptic elements. Other neurotransmitter abnormalities, however, have been observed in this and other basal ganglia diseases. This chapter wi

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Pyridine Toxins,g on experimental models, would be flawed without a discussion of this newly discovered relationship. This striking turn of events has been precipitated almost entirely by the discovery of a single compound, l-methyl-4-phenyl-l,2,3,6-tetrahydropyridine, or, as it is now widely know, MPTP. In this ch