Titlebook: Drug Resistance in Leukemia and Lymphoma III; G. J. L. Kaspers,R. Pieters,A. J. P. Veerman Book 1999 The Editor(s) (if applicable) and The

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micronized

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https://doi.org/10.1007/978-3-322-85040-9ence (NF), is related to cell compounds, i.e. proteins (1), nicotinic coenzymes (2), flavins (3), etc., engaged in structural and metabolic processes. Thus a correlation does exist between NF emission and morpho-functional state of cells.

Subjugate

Discrete

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國(guó)家明智

Multidrug Resistance Related Proteins in Primary Cutaneous Lymphomas then polychemotherapy is indicated. Multi-agent chemotherapy may induce long lasting complete remissions in CBCL’s. Most CTCL’s, especially mycosis fungoides (MF), and CD30 negative primary cutaneous large T-cell lymphoma (PCLTCL) respond poorly or partially to Multi-agent Chemotherapy.

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Book 1999e protein), and the role of glutathione and glutathione-S-transferases. Moreover, more drug type-specific mechanisms of resistance are a topic, such as for glucocorticoids and antifolates. Much in- formation is provided on apoptosis and its regulators, and on the results of cell culture drug resista

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https://doi.org/10.1007/978-94-015-9325-0ug resistance: (i) using multivariate analysis, MDR1 phenotype appears to be an independent variable, as potent (or higher) as karyo-type and age for predicting . drug resistance; (ii) the prognostic value is limited to the CD34(+)/P-gp(+) phenotype, wich is linked to a functional P-gp; (iii) the .

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