Titlebook: Dopamine and Glutamate in Psychiatric Disorders; Werner J. Schmidt,Maarten E. A. Reith Book 2005 Humana Press 2005 Alzheimer.Parkinson.att

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https://doi.org/10.1007/978-1-59259-852-6Alzheimer; Parkinson; attention; attention deficit hyperactivity disorder (ADHD); biochemistry; dementia;

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https://doi.org/10.1007/978-3-658-09108-8e affinity of antipsychotic drugs for dopamine receptors is highly correlated with clinical potency (.,.), and that the density of [.H]neuroleptic-labeled dopamine receptors is enhanced in postmortem brain tissue of schizophrenics (.), the study of dopamine receptors has been inextricably linked wit

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Ungleichheitssensible Hochschullehre known since 1978 to be divided between two families differing in biochemical and pharmacological properties (.). Although the G protein and second messenger systems affected by dopamine receptors in vivo have not been clearly established, in vitro D1-family receptors (D1 and D5) couple to Gs stimul

脆弱吧

René; Breiwe,Anke Liegmann,Stephan Ottoonotropic and metabotropic receptors abundantly expressed along the whole extent of the neuraxis. Abnormal regulation of glutamatergic transmission is, therefore, a key factor that underlies the appearance and progression of many neurodegenerative and psychiatric diseases. Unfortunately, the success

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https://doi.org/10.1007/978-3-658-10223-4ors that includes .-methyl-.-aspartate (NMDA), α-amino-3-hydroxy-5-methylisoxazole-4-propionate (AMPA), and kainate receptors, and (.) a group of metabotropic receptors (mGluRs). Ionotropic glutamate receptors, which are ligand-gated ion channels permeable for Ca., Na., and K., are responsible for f

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What Drives Elite-Challenging Behaviours?ly grounded on the principle of “reverse engineering.” In this case, the demonstrable effects of “first generation” antidepressants (e.g., tricyclics, such as imipramine) on the reuptake of norepinephrine and serotonin (.,.), and the observation that drugs depleting these biogenic amines lower mood