Titlebook: Disorders of Porphyrin Metabolism; Michael R. Moore,Kenneth E. L. McColl,Abraham Gold Book 1987 Springer Science+Business Media New York 1

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Perspective,gical and the medico—chemical.” The first goes back at least a billion years, but is outside the scope of this summary. The latter began in the early 1840s with chemical treatment of hemoglobin with concentrated sulfuric acid. In this way so-called “iron-free hematin” was formed. The tetrapyrrolic m

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https://doi.org/10.1007/978-1-4684-1277-2chemistry; classification; development; management; metabolism

cardiovascular

978-1-4684-1279-6Springer Science+Business Media New York 1987

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Joachim Dahm,Rolfjosef HamacherThe acute porphyrias are a group of genetic diseases characterized biochemically by excessive excretion of heme precursors and clinically by episodes of neurovisceral dysfunction and in some forms also by cutaneous photosensitivity.

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,Die Deutsche Terminb?rse (DTB),idges (Fig. 7). This is a rigid planar structure onto which eight side chains can be attached at positions 1 to 8 in the Fischer nomenclature. The type of side chain determines the physical characteristics of the porphyrin. The four pyrrole rings are designated A, B, C, and D and the four methine br

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Der hierarchisch ablaufende Planungsprozess,ric manifestations. This symptomatology can be explained almost entirely on a neurogenic basis. There is currently a fairly complete understanding of both genetic and biochemical factors in acute porphyria, but despite this the relationship between these clinical features and the biochemical present

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Rihard Karba,Ju? Kocijan,Gorazd Karerh century. This produced the first example of acute porphyria (Stokvis, 1889) and was followed by a number of similar cases. Since that time, a number of commonly used drugs have been linked with the development of acute attacks of these diseases. Workers have used the ability of certain compounds t