Titlebook: Diffuse Low-Grade Gliomas in Adults; Hugues Duffau Book 2017Latest edition Springer-Verlag London Ltd. 2017 Gliomagenesis.DLGG.neuro-oncol

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Epidemiology of Diffuse Low Grade Gliomasnt of surgical resection, histology, biology, etc.) are discussed and it is shown how they influence survival. Recent literature proposes a lot of spontaneous prognostic factors, but until now, just a few are validated. On the other hand, little data are available to define best combinations of the

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Molecular-Genetic Classification of Gliomas and Its Practical Application to Diagnostic Neuropatholof adults, as well as secondary GBMs, and are a major discriminate of biologic class. .-mutant diffusely infiltrative astrocytomas (grades II and III), as well as secondary GBMs, are characterized by . and . mutations. Oligodendrogliomas are also .-mutant, but instead are characterized by 1p/19q co-d

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Towards an Intermediate Grade in Glioma Classificationuld benefit from early adjuvant treatments. We have studied the heterogeneity within GII gliomas, and find that some tumors harbor foci with histopathological and molecular features similar to higher grade. We suggest that these foci may represent initiation of malignant transformation. The term GII

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Foreshadow

Diffuse Low-Grade Glioma Associated Stem Cellsf the tumor microenvironment. Importantly, GASC resulted to be the strongest predictors of LGG patients’ overall survival and malignant progression free survival, outperforming the state-of-the-art prognostic factors, including IDH1/2 mutation and 1p/19q co-deletion..In this chapter, we will first b

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Molecular Imaging of Diffuse Low Grade Gliomad FDOPA provide additional avenues of tumor characterization that are unfettered by the high background uptake of FDG. The future may be characterized by gains in predictive, rather than merely prognostic markers, that could help optimize patient outcomes. As more targeted therapies become available

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Clinical Presentation in Diffuse Low-Grade Gliomasatic”. These insidious symptoms may be unrecognized but present and affect emotional and cognitive functions. Also, malignant tumor transformation can precede the development of clinical symptoms for long periods of time, indicating that the absence of progressive symptoms does not protect against m

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Epilepsy and Diffuse Low-Grade Gliomasltrated by sparse glioma cells and glioma-related epileptogenic mechanisms are multifactorial and intermixed. An excessive glutamatergic excitatory neurotransmission is induced by a high extracellular glutamate concentration resulting from a decrease in glutamate uptake and from an increase in gluta

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Positron-Emission-Tomography in Diffuse Low-Grade Gliomastherapy. In the further course of the disease, amino acid PET allows a sensitive monitoring of treatment response, the early detection of tumor recurrence, and an improved differentiation of tumor recurrence from treatment-related changes. In the past, the method had only limited availability due to