Titlebook: Dietary Phenylalanine and Brain Function; Richard J. Wurtman,Eva Ritter-Walker Book 1988 Birkh?user Boston 1988 brain.metabolism.perceptio

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Overview: 978-1-4615-9823-7978-1-4615-9821-3

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Caroline Richter,Katharina Mojescikvalues are statistically significant. Latency time and recovery time were not significantly changed. Orally administered doses of aspartame up to 600 mg/kg did not change the 50% convulsion dose of lidocaine.

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Konventionen und Normen der Produktqualit?ts or milk provide around 5 g of phenylalanine (Phe) per 100 g of protein. A large fraction of dietary Phe is hydroxylated to tyrosine in the liver, a process catalyzed by the enzyme Phe-hydroxylase. Plasma Phe levels, like those of the other amino acids, are not regulated, and at any given time they

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Einführung in das Qualit?tsmanagementhese clinical studies have examined the plasma phenylalanine response when APM was given as a bolus dose in unsweetened beverage, orange juice, a sucrose solution, capsules, or a slurry or as part of a meal. The complexity of the meal feedings ranged from soup and a beverage to a hamburger and a mil

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Beeintr?chtigungen der Sprachentwicklung test followed by chronic intake for a period of 12 weeks. Two doses of aspartame were used in the loading test, 50 mg/kg and 100 mg/kg. Blood levels of phenylalanine and tyrosine were measured hourly, and the aromatic acid metabolites of phenylalanine were determined in urine. During the chronic in