Titlebook: Diagnosis and Management of Polycystic Ovary Syndrome; Evanthia Diamanti-Kandarakis,NADIR R. FARID Book 2009 Springer-Verlag US 2009 Diabe

Halfhearted

Omniscient

Obligatory

Amnesty

Insulin Resistance in PCOSincreased amounts of insulin secreted in the circulation. However, this is only an approximate description of this disorder. Insulin resistance encompasses an elaborate clinical, pathophysiologic, and molecular spectrum, and therefore, a well-established definition remains elusive.

沉思的魚

Genetics of Metabolic Syndrome and Genetic Lipodystrophiestance, type 2 diabetes (DM2), and dyslipidemia [1–3]. Although the presence of interfamilial and intrafamilial variation in the phenotype of PCOS-affected individuals points to an important role for environmental influences, studies demonstrating familial aggregation of PCOS and its associated metab

commensurate

The Genetics of Polycystic Ovary Syndrome by chronic anovulation, hyperandrogenemia, and often obesity [1]. Key reproductive features of PCOS are disordered gonadotropin secretion [2], elevated ovarian and adrenal androgen production [3], and frequently polycystic ovaries. Women with PCOS have profound insulin resistance as well as beta ce

Obverse

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