Titlebook: Developmental Neuropathology; Reinhard L. Friede Book 1989Latest edition Springer-Verlag Berlin Heidelberg 1989 Hydrocephalus.aneurysm.bra

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書目名稱Developmental Neuropathology影響因子(影響力)




書目名稱Developmental Neuropathology影響因子(影響力)學(xué)科排名




書目名稱Developmental Neuropathology網(wǎng)絡(luò)公開度




書目名稱Developmental Neuropathology網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Developmental Neuropathology被引頻次




書目名稱Developmental Neuropathology被引頻次學(xué)科排名




書目名稱Developmental Neuropathology年度引用




書目名稱Developmental Neuropathology年度引用學(xué)科排名




書目名稱Developmental Neuropathology讀者反饋




書目名稱Developmental Neuropathology讀者反饋學(xué)科排名

含糊

托運(yùn)

Porencephaly, Hydranencephaly, Multicystic Encephalopathytissue during early development. There is inconsistency in the naming of these lesions. In neuroradiology, it is customary to use the term porencephaly indiscriminately for all large cavities in the brains of infants. Neuropathologic features allow a more detailed classification: hydranencephaly, ba

Terminal

Hemorrhages in Asphyxiated Premature Infantsins of asphyxiated premature infants. Clinical studies using ultrasonography or CT show an incidence of germinal layer/intraventricular hemorrhage as high as 43 and 46% in infants below 1500 g birth weight (Hawgood et al. 1984); however, there are considerable differences between frequencies reporte

嬉耍

憤怒事實(shí)

Perinatal Lesions of White Matterto the Obstetric Society of London in 1861, following two earlier publications (1843, 1853), reviewed his experience with 200 patients, 63 of them specified in a table. All had had prolonged labor, instrumental delivery or severe respiratory difficulties at birth and all suffered from disturbances o

憤怒事實(shí)

社團(tuán)

Postnatal Lesions of Gray Matter. 7. Several of the “perinatal” lesions described in the previous chapter can also develop in early infancy, e. g. status marmoratus or ulegyria. Conversely, cardiac arrest encephalopathy, described in this chapter, was often reported as a typical form of perinatal damage. None the less, on review o

legislate

Kernicterus (Bilirubin Encephalopathy) and the ventricular walls. The first comprehensive description of the disease was given by Schmorl (1903), who distinguished two types of cerebral lesions in icteric infants. One was a diffuse staining of the tissue in infants with periventricular infarcts. The other lesion, called kernicterus by S

LURE

Arterial Diseases in Infancyystic encephalopathy described in Chap. 3. Porencephalies and hydranencephalies develop during fetal life, often bilateral and in middle cerebral artery distribution. They cause smooth-walled, full thickness defects, often with developmental anomalies in the adjacent cortex. Cerebral infarcts during