Titlebook: Development of the Cerebellum from Molecular Aspects to Diseases; Hassan Marzban Book 20171st edition Springer International Publishing AG

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The Development of the Cerebellum: From the Beginnings,e late nineteenth century until the late 1970s provided substantial and significant information; however, it was only descriptive and barely addressed the mechanisms involved.” Observations and their description, the nomenclature that evolved from these studies, and the ideas they fostered, indeed,

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Cellular and Genetic Programs Underlying Cerebellum Development,he brain in a complex set of folds. To accommodate production of the large number of cells, the cerebellum has not only a ventricular progenitor zone that produces all the glia and inhibitory neurons but also a unique progenitor zone, the rhombic lip, dedicated to excitatory neuron production. In th

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Early Purkinje Cell Development and the Origins of Cerebellar Patterning, control of PC progenitor specification, proliferation, subtype differentiation, migration, and survival from the cerebellar primordium to the end of prenatal embryogenesis, discussing some of the key molecules involved and the ways they combine to generate the complex adult cerebellar architecture.

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Motor Circuit Abnormalities During Cerebellar Development,xia, dystonia, and tremor. Cerebellar connectivity in both normal and abnormal states has been intensely studied. As a result, its anatomy, circuitry, and neuronal firing properties are among the best understood in the brain. This knowledge has directly facilitated efforts to uncover the mechanisms

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Developmental Disorders of the Cerebellum and Neurotrophic Factors,lts in neurological disorders such as attention deficit hyperactivity disorder (ADHD), congenital ataxia, and autism. Because neurotrophic factors have established effects on the growth, proliferation, differentiation, and arborization of neurons, their role in the neurodevelopmental disorders has b

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Apoptosis, Autophagy, and Unfolded Protein Response and Cerebellar Development,development. Among these mechanisms, apoptosis, autophagy, and unfolded protein response play critical roles in regulation of development by affecting the cell fate. All of these pathways are involved in regulation of cell number via determining the life and death cycles of the cells. In this chapte

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The Ubiquitin Proteasome System and Cerebellar Developmental Disease,. Dysfunction of the UPS is observed in several types of spinocerebellar ataxias associated with polyglutamine accumulation. Spinocerebellar ataxia type 3 is caused by a genetic defect ion the Atxn3 gene, which codes for a deubiquitinase enzyme. Defects in expression of a variety of ubiquitin ligase